Lucia Schwarzfischer
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View article: Supplementary Figure 6 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
Supplementary Figure 6 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia Open
Supplementary Figure 6 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
View article: Supplementary Figure 3 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
Supplementary Figure 3 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia Open
Supplementary Figure 3 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
View article: Supplementary Table 4 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
Supplementary Table 4 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
Supplementary Table 4 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells
View article: Supplementary Figure 5 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
Supplementary Figure 5 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia Open
Supplementary Figure 5 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
View article: TrackEpityper Data THP1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
TrackEpityper Data THP1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
TrackEpityper Data THP1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells
View article: Supplementary Figure 5 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
Supplementary Figure 5 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia Open
Supplementary Figure 5 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
View article: Supplementary Figure 6 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
Supplementary Figure 6 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia Open
Supplementary Figure 6 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
View article: Data from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
Data from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia Open
The methylation of CpG islands is associated with transcriptional repression and, in cancer, leads to the abnormal silencing of tumor suppressor genes. Because aberrant hypermethylation may be used as a marker for disease, a sensitive meth…
View article: TrackCHIP Mono NRF1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
TrackCHIP Mono NRF1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
TrackCHIP Mono NRF1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells
View article: TrackCHIP THP1vsMono from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
TrackCHIP THP1vsMono from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
TrackCHIP THP1vsMono from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells
View article: TrackEpityper Data U937 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
TrackEpityper Data U937 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
TrackEpityper Data U937 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells
View article: Data from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
Data from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
Aberrant DNA methylation at CpG islands is thought to contribute to cancer initiation and progression, but mechanisms that establish and maintain DNA methylation status during tumorigenesis or normal development remain poorly understood. I…
View article: Supplementary Figure 4 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
Supplementary Figure 4 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia Open
Supplementary Figure 4 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
View article: Supplementary Figure 1 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
Supplementary Figure 1 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia Open
Supplementary Figure 1 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
View article: TrackCHIP Mono Sp1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
TrackCHIP Mono Sp1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
TrackCHIP Mono Sp1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells
View article: Supplementary Table 4 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
Supplementary Table 4 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
Supplementary Table 4 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells
View article: Supplementary Table 2 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
Supplementary Table 2 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
Supplementary Table 2 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells
View article: Supplementary Figure 2 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
Supplementary Figure 2 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia Open
Supplementary Figure 2 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
View article: Data from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
Data from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
Aberrant DNA methylation at CpG islands is thought to contribute to cancer initiation and progression, but mechanisms that establish and maintain DNA methylation status during tumorigenesis or normal development remain poorly understood. I…
View article: TrackCHIP Mono YY1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
TrackCHIP Mono YY1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
TrackCHIP Mono YY1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells
View article: Supplementary Materials and Methods, Figures 1-7 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
Supplementary Materials and Methods, Figures 1-7 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
Supplementary Materials and Methods, Figures 1-7 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells
View article: TrackEpityper Data U937 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
TrackEpityper Data U937 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
TrackEpityper Data U937 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells
View article: Supplementary Figure 2 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
Supplementary Figure 2 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia Open
Supplementary Figure 2 from Genome-Wide Profiling of CpG Methylation Identifies Novel Targets of Aberrant Hypermethylation in Myeloid Leukemia
View article: Supplementary Table 1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
Supplementary Table 1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
Supplementary Table 1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells
View article: TrackEpityper Data Mono from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
TrackEpityper Data Mono from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
TrackEpityper Data Mono from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells
View article: TrackCHIP U937vsMono from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
TrackCHIP U937vsMono from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
TrackCHIP U937vsMono from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells
View article: TrackCHIP Mono Sp1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
TrackCHIP Mono Sp1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
TrackCHIP Mono Sp1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells
View article: TrackEpityper Amplicons from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
TrackEpityper Amplicons from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
TrackEpityper Amplicons from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells
View article: TrackCHIP Mono YY1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
TrackCHIP Mono YY1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
TrackCHIP Mono YY1 from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells
View article: TrackCHIP THP1vsMono from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells
TrackCHIP THP1vsMono from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to <i>De novo</i> DNA Methylation in Cancer Cells Open
TrackCHIP THP1vsMono from General Transcription Factor Binding at CpG Islands in Normal Cells Correlates with Resistance to De novo DNA Methylation in Cancer Cells