Marc Guiu
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View article: A bioinformatics screen identifies <scp>TCF19</scp> as an aggressiveness‐sustaining gene in prostate cancer
A bioinformatics screen identifies <span>TCF19</span> as an aggressiveness‐sustaining gene in prostate cancer Open
Prostate cancer is a prevalent tumor type that, despite being highly curable, progresses to metastatic disease in a fraction of patients, thus accounting for more than 350 000 annual deaths worldwide. In turn, uncovering the molecular insi…
View article: MAF amplification licenses ERα through epigenetic remodelling to drive breast cancer metastasis
MAF amplification licenses ERα through epigenetic remodelling to drive breast cancer metastasis Open
MAF amplification increases the risk of breast cancer (BCa) metastasis through mechanisms that are still poorly understood yet have important clinical implications. Oestrogen-receptor-positive (ER + ) BCa requires oestrogen for both growth…
View article: Supplementary Figures and Tables from ERK1/2 Signaling Induces Upregulation of ANGPT2 and CXCR4 to Mediate Liver Metastasis in Colon Cancer
Supplementary Figures and Tables from ERK1/2 Signaling Induces Upregulation of ANGPT2 and CXCR4 to Mediate Liver Metastasis in Colon Cancer Open
SF1: The expression of ANGPT2 and CXCR4 in CRC; SF2: Expression levels of ANGPT2, CXCR4, CXCL12, TEK and VEGFA in SW620_P and SW620_LiM2 cells; SF3: Expression levels of ANGPT2 and CXCR4 upon downregulation and upregulation.; SF4: CXCL1 an…
View article: Supplementary Materials and Methods from ERK1/2 Signaling Induces Upregulation of ANGPT2 and CXCR4 to Mediate Liver Metastasis in Colon Cancer
Supplementary Materials and Methods from ERK1/2 Signaling Induces Upregulation of ANGPT2 and CXCR4 to Mediate Liver Metastasis in Colon Cancer Open
Supplementary Materials and Methods
View article: Supplementary Figures and Tables from ERK1/2 Signaling Induces Upregulation of ANGPT2 and CXCR4 to Mediate Liver Metastasis in Colon Cancer
Supplementary Figures and Tables from ERK1/2 Signaling Induces Upregulation of ANGPT2 and CXCR4 to Mediate Liver Metastasis in Colon Cancer Open
SF1: The expression of ANGPT2 and CXCR4 in CRC; SF2: Expression levels of ANGPT2, CXCR4, CXCL12, TEK and VEGFA in SW620_P and SW620_LiM2 cells; SF3: Expression levels of ANGPT2 and CXCR4 upon downregulation and upregulation.; SF4: CXCL1 an…
View article: Supplementary Materials and Methods from ERK1/2 Signaling Induces Upregulation of ANGPT2 and CXCR4 to Mediate Liver Metastasis in Colon Cancer
Supplementary Materials and Methods from ERK1/2 Signaling Induces Upregulation of ANGPT2 and CXCR4 to Mediate Liver Metastasis in Colon Cancer Open
Supplementary Materials and Methods
View article: Data from ERK1/2 Signaling Induces Upregulation of ANGPT2 and CXCR4 to Mediate Liver Metastasis in Colon Cancer
Data from ERK1/2 Signaling Induces Upregulation of ANGPT2 and CXCR4 to Mediate Liver Metastasis in Colon Cancer Open
Carcinoma development in colorectal cancer is driven by genetic alterations in numerous signaling pathways. Alterations in the RAS-ERK1/2 pathway are associated with the shortest overall survival for patients after diagnosis of colorectal …
View article: Data from ERK1/2 Signaling Induces Upregulation of ANGPT2 and CXCR4 to Mediate Liver Metastasis in Colon Cancer
Data from ERK1/2 Signaling Induces Upregulation of ANGPT2 and CXCR4 to Mediate Liver Metastasis in Colon Cancer Open
Carcinoma development in colorectal cancer is driven by genetic alterations in numerous signaling pathways. Alterations in the RAS-ERK1/2 pathway are associated with the shortest overall survival for patients after diagnosis of colorectal …
View article: Supplementary Tables 1 - 3 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence
Supplementary Tables 1 - 3 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence Open
XLSX file - 225K, Log2FC of the 1631 gene transcripts regulated in MCF7 Tet-Off/p95HER2 60 hours after doxycycline removal and log2FC of the same transcripts in MCF7 Tet-Off/HER2 cells under the same conditions; Log2FC of the 361 genes pro…
View article: Supplementary Tables 1 - 3 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence
Supplementary Tables 1 - 3 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence Open
XLSX file - 225K, Log2FC of the 1631 gene transcripts regulated in MCF7 Tet-Off/p95HER2 60 hours after doxycycline removal and log2FC of the same transcripts in MCF7 Tet-Off/HER2 cells under the same conditions; Log2FC of the 361 genes pro…
View article: Supplementary Figure 3 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence
Supplementary Figure 3 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence Open
PDF file - 62K, The tyrosine kinase inhibitor lapatinib does not revert the cellular senescence induced by p95HER2
View article: Supplementary Methods, Figures 1-10, Table 1 from HER2 Silences Tumor Suppression in Breast Cancer Cells by Switching Expression of C/EBPβ Isoforms
Supplementary Methods, Figures 1-10, Table 1 from HER2 Silences Tumor Suppression in Breast Cancer Cells by Switching Expression of C/EBPβ Isoforms Open
Supplementary Methods, Figures 1-10, Table 1 from HER2 Silences Tumor Suppression in Breast Cancer Cells by Switching Expression of C/EBPβ Isoforms
View article: Supplementary Figure 4 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence
Supplementary Figure 4 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence Open
PDF file - 136K, Expression of p95HER2 leads to senescence in MCF7 cell xenografts
View article: Supplementary Figure 5 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence
Supplementary Figure 5 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence Open
PDF file - 53K, The removal of doxycycline from the drinking water does not affect the metastatic growth of MDA-MB-231 cells
View article: Supplementary Figure Legends 1-6 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence
Supplementary Figure Legends 1-6 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence Open
PDF file - 76K
View article: Supplementary Figure 4 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence
Supplementary Figure 4 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence Open
PDF file - 136K, Expression of p95HER2 leads to senescence in MCF7 cell xenografts
View article: Supplementary Figure 5 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence
Supplementary Figure 5 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence Open
PDF file - 53K, The removal of doxycycline from the drinking water does not affect the metastatic growth of MDA-MB-231 cells
View article: Supplementary Figure 1 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence
Supplementary Figure 1 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence Open
PDF file - 104K, Expression of p95HER2 in MCF10A, T47D and MDA-MB-453 cells and expression of HER2 in MCF7 cells
View article: Data from HER2 Silences Tumor Suppression in Breast Cancer Cells by Switching Expression of C/EBPβ Isoforms
Data from HER2 Silences Tumor Suppression in Breast Cancer Cells by Switching Expression of C/EBPβ Isoforms Open
Tumor progression requires ablation of suppressor functions mediated by transforming growth factor β (TGFβ) signaling and by oncogene-induced senescence (OIS), but how these functions are canceled in specific subtypes of breast cancer rema…
View article: Data from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence
Data from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence Open
Senescence, a terminal cell proliferation arrest, can be triggered by oncogenes. Oncogene-induced senescence is classically considered a tumor defense barrier. However, several findings show that, under certain circumstances, senescent cel…
View article: Supplementary Figure 6 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence
Supplementary Figure 6 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence Open
PDF file - 65K, Growth of a mixture 1:1 of MCF7 Tet-off / p95HER2 and MDA-MB-231 / Luc cells in the presence or absence of doxycycline
View article: Supplementary Figure 6 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence
Supplementary Figure 6 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence Open
PDF file - 65K, Growth of a mixture 1:1 of MCF7 Tet-off / p95HER2 and MDA-MB-231 / Luc cells in the presence or absence of doxycycline
View article: Supplementary Figure 2 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence
Supplementary Figure 2 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence Open
PDF file - 54K, Metabolic activity of p95HER2-induced senescent cells
View article: Data from HER2 Silences Tumor Suppression in Breast Cancer Cells by Switching Expression of C/EBPβ Isoforms
Data from HER2 Silences Tumor Suppression in Breast Cancer Cells by Switching Expression of C/EBPβ Isoforms Open
Tumor progression requires ablation of suppressor functions mediated by transforming growth factor β (TGFβ) signaling and by oncogene-induced senescence (OIS), but how these functions are canceled in specific subtypes of breast cancer rema…
View article: Data from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence
Data from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence Open
Senescence, a terminal cell proliferation arrest, can be triggered by oncogenes. Oncogene-induced senescence is classically considered a tumor defense barrier. However, several findings show that, under certain circumstances, senescent cel…
View article: Supplementary Figure Legends 1-6 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence
Supplementary Figure Legends 1-6 from Constitutive HER2 Signaling Promotes Breast Cancer Metastasis through Cellular Senescence Open
PDF file - 76K
View article: Supplementary Methods, Figures 1-10, Table 1 from HER2 Silences Tumor Suppression in Breast Cancer Cells by Switching Expression of C/EBPβ Isoforms
Supplementary Methods, Figures 1-10, Table 1 from HER2 Silences Tumor Suppression in Breast Cancer Cells by Switching Expression of C/EBPβ Isoforms Open
Supplementary Methods, Figures 1-10, Table 1 from HER2 Silences Tumor Suppression in Breast Cancer Cells by Switching Expression of C/EBPβ Isoforms