Mark Kerber
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View article: Impaired mitochondrial metabolism is a critical cancer vulnerability for MYC inhibitors
Impaired mitochondrial metabolism is a critical cancer vulnerability for MYC inhibitors Open
MYC is a key driver in many aggressive and therapy-resistant cancers. We have developed and characterized a small-molecule MYC inhibitor named MYCi975. To uncover combination strategies for MYC inhibitors, we conducted a genome-wide CRISPR…
View article: Supplementary Figure 2a from Flt-1 Signaling in Macrophages Promotes Glioma Growth <i>In vivo</i>
Supplementary Figure 2a from Flt-1 Signaling in Macrophages Promotes Glioma Growth <i>In vivo</i> Open
Supplementary Figure 2a from Flt-1 Signaling in Macrophages Promotes Glioma Growth In vivo
View article: Supplementary Figure 2b from Flt-1 Signaling in Macrophages Promotes Glioma Growth <i>In vivo</i>
Supplementary Figure 2b from Flt-1 Signaling in Macrophages Promotes Glioma Growth <i>In vivo</i> Open
Supplementary Figure 2b from Flt-1 Signaling in Macrophages Promotes Glioma Growth In vivo
View article: Data from Flt-1 Signaling in Macrophages Promotes Glioma Growth <i>In vivo</i>
Data from Flt-1 Signaling in Macrophages Promotes Glioma Growth <i>In vivo</i> Open
Several lines of evidence indicate that Flt-1, a fms-like tyrosine kinase receptor, which binds to vascular endothelial growth factor (VEGF)-A, VEGF-B, and PlGF, is a positive regulator of angiogenesis in the context of tumor growth and me…
View article: Supplementary Figure 1 from Flt-1 Signaling in Macrophages Promotes Glioma Growth <i>In vivo</i>
Supplementary Figure 1 from Flt-1 Signaling in Macrophages Promotes Glioma Growth <i>In vivo</i> Open
Supplementary Figure 1 from Flt-1 Signaling in Macrophages Promotes Glioma Growth In vivo
View article: Data from Flt-1 Signaling in Macrophages Promotes Glioma Growth <i>In vivo</i>
Data from Flt-1 Signaling in Macrophages Promotes Glioma Growth <i>In vivo</i> Open
Several lines of evidence indicate that Flt-1, a fms-like tyrosine kinase receptor, which binds to vascular endothelial growth factor (VEGF)-A, VEGF-B, and PlGF, is a positive regulator of angiogenesis in the context of tumor growth and me…
View article: Supplementary Figure 1 from Flt-1 Signaling in Macrophages Promotes Glioma Growth <i>In vivo</i>
Supplementary Figure 1 from Flt-1 Signaling in Macrophages Promotes Glioma Growth <i>In vivo</i> Open
Supplementary Figure 1 from Flt-1 Signaling in Macrophages Promotes Glioma Growth In vivo
View article: Supplementary Figure 2b from Flt-1 Signaling in Macrophages Promotes Glioma Growth <i>In vivo</i>
Supplementary Figure 2b from Flt-1 Signaling in Macrophages Promotes Glioma Growth <i>In vivo</i> Open
Supplementary Figure 2b from Flt-1 Signaling in Macrophages Promotes Glioma Growth In vivo
View article: Supplementary Figure 2a from Flt-1 Signaling in Macrophages Promotes Glioma Growth <i>In vivo</i>
Supplementary Figure 2a from Flt-1 Signaling in Macrophages Promotes Glioma Growth <i>In vivo</i> Open
Supplementary Figure 2a from Flt-1 Signaling in Macrophages Promotes Glioma Growth In vivo