Mei‐Hui Lin
YOU?
Author Swipe
View article: Defects in energy metabolism increase the susceptibility of <i>Staphylococcus aureus</i> and its small colony variants (SCVs) to <i>Staphylococcus lugdunensis</i> and lugdunin
Defects in energy metabolism increase the susceptibility of <i>Staphylococcus aureus</i> and its small colony variants (SCVs) to <i>Staphylococcus lugdunensis</i> and lugdunin Open
Lugdunin, produced by Staphylococcus lugdunensis , exhibits potent antimicrobial activity against a broad range of Gram-positive bacteria, including Staphylococcus aureus . This study aims to elucidate the mechanisms underlying the suscept…
View article: Staphylococcus aureus foldase PrsA contributes to the folding and secretion of protein A
Staphylococcus aureus foldase PrsA contributes to the folding and secretion of protein A Open
View article: The in vivo biofilm formation of Staphylococcus aureus in a mouse model: albumin is the major biofilm matrix
The in vivo biofilm formation of Staphylococcus aureus in a mouse model: albumin is the major biofilm matrix Open
Generally, Staphylococcus aureus biofilms are composed of proteins, extracellular DNA (eDNA), and polysaccharide intercellular adhesin (PIA). Our knowledge of biofilm development is largely derived from in vitro systems. The molecular mech…
View article: First in human data of NKX019, an allogeneic CAR NK for the treatment of relapsed/refractory (R/R) B‐cell malignancies
First in human data of NKX019, an allogeneic CAR NK for the treatment of relapsed/refractory (R/R) B‐cell malignancies Open
Background: Autologous CAR T-cell therapies have altered the treatment landscape for many patients (pts) with advanced B-cell malignancies, however custom manufacturing precludes prompt treatment and can result in manufacturing failure. T-…
View article: Supplementary Methods and References from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models
Supplementary Methods and References from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models Open
Supplementary Methods and References from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy In vivo in Human Acute Myeloid Leukemia Models
View article: Supplementary Figure Legends from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models
Supplementary Figure Legends from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models Open
Supplementary Figure Legends from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy In vivo in Human Acute Myeloid Leukemia Models
View article: Supplementary Methods and References from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models
Supplementary Methods and References from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models Open
Supplementary Methods and References from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy In vivo in Human Acute Myeloid Leukemia Models
View article: Supplementary Figures 7-8 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models
Supplementary Figures 7-8 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models Open
Supplementary Figures 7-8 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy In vivo in Human Acute Myeloid Leukemia Models
View article: Data from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models
Data from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models Open
We examined whether potent vascular endothelial growth factor (VEGF) blockade mediated by aflibercept, a decoy VEGF receptor (VEGFR) 1/2 moiety with stronger affinity for VEGF than bevacizumab, resulted in antileukemia effects and enhanced…
View article: Supplementary Figures 1-6 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models
Supplementary Figures 1-6 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models Open
Supplementary Figures 1-6 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy In vivo in Human Acute Myeloid Leukemia Models
View article: Supplementary Figures 1-6 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models
Supplementary Figures 1-6 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models Open
Supplementary Figures 1-6 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy In vivo in Human Acute Myeloid Leukemia Models
View article: Supplementary Tables 1-3 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models
Supplementary Tables 1-3 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models Open
Supplementary Tables 1-3 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy In vivo in Human Acute Myeloid Leukemia Models
View article: Supplementary Tables 1-3 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models
Supplementary Tables 1-3 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models Open
Supplementary Tables 1-3 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy In vivo in Human Acute Myeloid Leukemia Models
View article: Supplementary Figures 7-8 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models
Supplementary Figures 7-8 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models Open
Supplementary Figures 7-8 from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy In vivo in Human Acute Myeloid Leukemia Models
View article: Data from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models
Data from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models Open
We examined whether potent vascular endothelial growth factor (VEGF) blockade mediated by aflibercept, a decoy VEGF receptor (VEGFR) 1/2 moiety with stronger affinity for VEGF than bevacizumab, resulted in antileukemia effects and enhanced…
View article: Supplementary Figure Legends from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models
Supplementary Figure Legends from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy <i>In vivo</i> in Human Acute Myeloid Leukemia Models Open
Supplementary Figure Legends from Aflibercept Exerts Antivascular Effects and Enhances Levels of Anthracycline Chemotherapy In vivo in Human Acute Myeloid Leukemia Models
View article: Hitchhiking motility of Staphylococcus aureus involves the interaction between its wall teichoic acids and lipopolysaccharide of Pseudomonas aeruginosa
Hitchhiking motility of Staphylococcus aureus involves the interaction between its wall teichoic acids and lipopolysaccharide of Pseudomonas aeruginosa Open
Staphylococcus aureus , which lacks pili and flagella, is nonmotile. However, it hitchhikes motile bacteria, such as Pseudomonas aeruginosa , to migrate in the environment. This study demonstrated that the hitchhiking motility of S. aureus…
View article: Relationship of symptom stress, care needs, social support, and meaning in life to quality of life in patients with heart failure from the acute to chronic stages: a longitudinal study
Relationship of symptom stress, care needs, social support, and meaning in life to quality of life in patients with heart failure from the acute to chronic stages: a longitudinal study Open
View article: Characteristics of HIV-positive pregnant women and HIV- and antiretroviral therapy-exposed fetuses: A case-control study
Characteristics of HIV-positive pregnant women and HIV- and antiretroviral therapy-exposed fetuses: A case-control study Open
Introduction: This study determined risk factors, obstetric comorbidities, and fetal conditions among HIV-positive mothers to improve their maternal care.
Methodology: This retrospective case-control study included HIV-positive pregnant w…
View article: Involvement of Heme in Colony Spreading of Staphylococcus aureus
Involvement of Heme in Colony Spreading of Staphylococcus aureus Open
Staphylococcus aureus spreads rapidly on the surface of soft agar medium. The spreading depends on the synthesis of biosurfactants, i.e., phenol soluble modulins (PSMs), which facilitate colony spreading of S. aureus. Our ear…
View article: <p>Grazoprevir/elbasvir in peginterferon alfa plus ribavirin experienced patients with chronic genotype 1 HCV/HIV co-infection: a non-randomized, open-label clinical trial</p>
Grazoprevir/elbasvir in peginterferon alfa plus ribavirin experienced patients with chronic genotype 1 HCV/HIV co-infection: a non-randomized, open-label clinical trial Open
Background and aims: We aimed to evaluate the efficacy and tolerability of grazoprevir/elbasvir in patients with chronic genotype 1 hepatitis C virus (HCV) and HIV co-infection who experienced peginterferon alfa plus ribavirin (PegI…
View article: The expression of fibronectin is significantly suppressed in macrophages to exert a protective effect against Staphylococcus aureus infection
The expression of fibronectin is significantly suppressed in macrophages to exert a protective effect against Staphylococcus aureus infection Open
The Src-JNK-c-Jun signaling pathway was activated in macrophages in response to S. aureus infection and resulted in the suppression of Fn expression. This suppression may play a protective role in macrophages against S. aureus infection. T…
View article: Modulation of Staphylococcus aureus spreading by water
Modulation of Staphylococcus aureus spreading by water Open
Staphylococcus aureus is known to spread rapidly and form giant colonies on the surface of soft agar and animal tissues by a process called colony spreading. So far, the mechanisms underlying spreading remain poorly understood. This study …
View article: High Glucose Concentration Promotes Vancomycin-Enhanced Biofilm Formation of Vancomycin-Non-Susceptible Staphylococcus aureus in Diabetic Mice
High Glucose Concentration Promotes Vancomycin-Enhanced Biofilm Formation of Vancomycin-Non-Susceptible Staphylococcus aureus in Diabetic Mice Open
We previously demonstrated that vancomycin treatment increased acquisition of eDNA and enhanced biofilm formation of drug-resistant Staphylococcus aureus through a cidA-mediated autolysis mechanism. Recently we found that such enhancement …
View article: Elucidating the Crucial Role of Poly N-Acetylglucosamine from Staphylococcus aureus in Cellular Adhesion and Pathogenesis
Elucidating the Crucial Role of Poly N-Acetylglucosamine from Staphylococcus aureus in Cellular Adhesion and Pathogenesis Open
Staphylococcus aureus is an important pathogen that forms biofilms on the surfaces of medical implants. Biofilm formation by S. aureus is associated with the production of poly N-acetylglucosamine (PNAG), also referred to as polysaccharide…