Nicolas Nottet
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View article: Figure S4 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness
Figure S4 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness Open
Lung micrometastasis
View article: Figure S5 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness
Figure S5 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness Open
PGC-1a, c-MYC and ODC1 expression in the Taylor database
View article: Figure S4 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness
Figure S4 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness Open
Lung micrometastasis
View article: Figure S3 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness
Figure S3 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness Open
The downregulation of ODC1 inhibits PCa aggressiveness in ShPGC-1a cells
View article: Supplementary Data from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness
Supplementary Data from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness Open
Legends to supplementary data
View article: Supplementary Data from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness
Supplementary Data from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness Open
Legends to supplementary data
View article: Data from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness
Data from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness Open
Although tumorigenesis is dependent on the reprogramming of cellular metabolism, the metabolic pathways engaged in the formation of metastases remain largely unknown. The transcriptional coactivator peroxisome proliferator-activated recept…
View article: Figure S2 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness
Figure S2 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness Open
PGC-a regulates c-MYC and ODC1 expression
View article: Figure S2 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness
Figure S2 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness Open
PGC-a regulates c-MYC and ODC1 expression
View article: Figure S1 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness
Figure S1 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness Open
PGC-1a suppresses PCa cell aggressiveness and polyamine synthesis
View article: Figure S1 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness
Figure S1 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness Open
PGC-1a suppresses PCa cell aggressiveness and polyamine synthesis
View article: Data from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness
Data from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness Open
Although tumorigenesis is dependent on the reprogramming of cellular metabolism, the metabolic pathways engaged in the formation of metastases remain largely unknown. The transcriptional coactivator peroxisome proliferator-activated recept…
View article: Figure S5 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness
Figure S5 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness Open
PGC-1a, c-MYC and ODC1 expression in the Taylor database
View article: Figure S3 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness
Figure S3 from PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness Open
The downregulation of ODC1 inhibits PCa aggressiveness in ShPGC-1a cells
View article: Tumoral microenvironment prevents de novo asparagine biosynthesis in B cell lymphoma, regardless of ASNS expression
Tumoral microenvironment prevents de novo asparagine biosynthesis in B cell lymphoma, regardless of ASNS expression Open
Depletion of circulating asparagine with l -asparaginase (ASNase) is a mainstay of leukemia treatment and is under investigation in many cancers. Expression levels of asparagine synthetase (ASNS), which catalyzes asparagine synthesis, were…
View article: CLEC12B Is a Melanocytic Gene Regulating the Color of the Skin
CLEC12B Is a Melanocytic Gene Regulating the Color of the Skin Open
View article: The FibromiR miR-214-3p Is Upregulated in Duchenne Muscular Dystrophy and Promotes Differentiation of Human Fibro-Adipogenic Muscle Progenitors
The FibromiR miR-214-3p Is Upregulated in Duchenne Muscular Dystrophy and Promotes Differentiation of Human Fibro-Adipogenic Muscle Progenitors Open
Fibrosis is a deleterious invasion of tissues associated with many pathological conditions, such as Duchenne muscular dystrophy (DMD) for which no cure is at present available for its prevention or its treatment. Fibro-adipogenic progenito…
View article: Correction: The nuclear hypoxia-regulated NLUCAT1 long non-coding RNA contributes to an aggressive phenotype in lung adenocarcinoma through regulation of oxidative stress
Correction: The nuclear hypoxia-regulated NLUCAT1 long non-coding RNA contributes to an aggressive phenotype in lung adenocarcinoma through regulation of oxidative stress Open
View article: Plk1, upregulated by HIF-2, mediates metastasis and drug resistance of clear cell renal cell carcinoma
Plk1, upregulated by HIF-2, mediates metastasis and drug resistance of clear cell renal cell carcinoma Open
Polo-like kinase 1 (Plk1) expression is inversely correlated with survival advantages in many cancers. However, molecular mechanisms that underlie Plk1 expression are poorly understood. Here, we uncover a hypoxia-regulated mechanism of Plk…
View article: Correction to: ITGBL1 is a new immunomodulator that favors development of melanoma tumors by inhibiting natural killer cells cytotoxicity
Correction to: ITGBL1 is a new immunomodulator that favors development of melanoma tumors by inhibiting natural killer cells cytotoxicity Open
View article: FBXO32 links ubiquitination to epigenetic reprograming of melanoma cells
FBXO32 links ubiquitination to epigenetic reprograming of melanoma cells Open
Ubiquitination by serving as a major degradation signal of proteins, but also by controlling protein functioning and localization, plays critical roles in most key cellular processes. Here, we show that MITF, the master transcription facto…
View article: Single-cell RNA sequencing reveals intratumoral heterogeneity in primary uveal melanomas and identifies HES6 as a driver of the metastatic disease
Single-cell RNA sequencing reveals intratumoral heterogeneity in primary uveal melanomas and identifies HES6 as a driver of the metastatic disease Open
View article: ITGBL1 is a new immunomodulator that favors development of melanoma tumors by inhibiting natural killer cells cytotoxicity
ITGBL1 is a new immunomodulator that favors development of melanoma tumors by inhibiting natural killer cells cytotoxicity Open
View article: Identification of oncolytic vaccinia restriction factors in canine high-grade mammary tumor cells using single-cell transcriptomics
Identification of oncolytic vaccinia restriction factors in canine high-grade mammary tumor cells using single-cell transcriptomics Open
Mammary carcinoma, including triple-negative breast carcinomas (TNBC) are tumor-types for which human and canine pathologies are closely related at the molecular level. The efficacy of an oncolytic vaccinia virus (VV) was compared in low-p…
View article: Identification of oncolytic vaccinia restriction factors in canine high-grade mammary tumor cells using single-cell transcriptomics
Identification of oncolytic vaccinia restriction factors in canine high-grade mammary tumor cells using single-cell transcriptomics Open
Mammary carcinoma, including triple-negative breast carcinomas (TNBC) are tumor-types for which human and canine pathologies are closely related at the molecular level. Low-passage, primary carcinoma cells from TNBC versus non-TNBC were us…
View article: Identification of a new aggressive axis driven by ciliogenesis and absence of VDAC1-ΔC in clear cell Renal Cell Carcinoma patients
Identification of a new aggressive axis driven by ciliogenesis and absence of VDAC1-ΔC in clear cell Renal Cell Carcinoma patients Open
Rationale: Renal cell carcinoma (RCC) accounts for about 2% of all adult cancers, and clear cell RCC (ccRCC) is the most common RCC histologic subtype. A hallmark of ccRCC is the loss of the primary cilium, a cellular antenna that s…
View article: PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness
PGC1α Inhibits Polyamine Synthesis to Suppress Prostate Cancer Aggressiveness Open
Although tumorigenesis is dependent on the reprogramming of cellular metabolism, the metabolic pathways engaged in the formation of metastases remain largely unknown. The transcriptional coactivator peroxisome proliferator-activated recept…
View article: A critical role for miR-142 in alveolar epithelial lineage formation in mouse lung development
A critical role for miR-142 in alveolar epithelial lineage formation in mouse lung development Open
View article: Lysosomal acid ceramidase ASAH1 controls the transition between invasive and proliferative phenotype in melanoma cells
Lysosomal acid ceramidase ASAH1 controls the transition between invasive and proliferative phenotype in melanoma cells Open
View article: Genome-wide Control of Heterochromatin Replication by the Telomere Capping Protein TRF2
Genome-wide Control of Heterochromatin Replication by the Telomere Capping Protein TRF2 Open