Oakleigh M. Folkes
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View article: Deficiency of Shank3 in the nucleus accumbens reveals a loss of social-specific motivation
Deficiency of Shank3 in the nucleus accumbens reveals a loss of social-specific motivation Open
Social behavior deficits are a hallmark of autism and other neuropsychiatric disorders. SHANK3, a common causal gene for autism, is highly expressed in the nucleus accumbens (NAc), a critical brain region for social behaviors. We previousl…
View article: Deficiency of <i>Shank3</i> in the Nucleus Accumbens Reveals a Loss of Social-Specific Motivation
Deficiency of <i>Shank3</i> in the Nucleus Accumbens Reveals a Loss of Social-Specific Motivation Open
Deficits in social interaction are a hallmark symptom of autism and other neuropsychiatric disorders. SHANK3 encodes a postsynaptic density scaffold protein and is one of the most common causal genes for autism. SHANK3 protein is highly ex…
View article: α2A-adrenergic heteroreceptors are required for stress-induced reinstatement of cocaine conditioned place preference
α2A-adrenergic heteroreceptors are required for stress-induced reinstatement of cocaine conditioned place preference Open
View article: An endocannabinoid-regulated basolateral amygdala–nucleus accumbens circuit modulates sociability
An endocannabinoid-regulated basolateral amygdala–nucleus accumbens circuit modulates sociability Open
Deficits in social interaction (SI) are a core symptom of autism spectrum disorders (ASDs); however, treatments for social deficits are notably lacking. Elucidating brain circuits and neuromodulatory signaling systems that regulate sociabi…
View article: Acute blockade of the Caenorhabditis elegans dopamine transporter DAT-1 by the mammalian norepinephrine transporter inhibitor nisoxetine reveals the influence of genetic modifications of dopamine signaling in vivo
Acute blockade of the Caenorhabditis elegans dopamine transporter DAT-1 by the mammalian norepinephrine transporter inhibitor nisoxetine reveals the influence of genetic modifications of dopamine signaling in vivo Open