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View article: Endomyocardial biopsy in clinical practice: the diagnostic yield and insights from a 5-year single-center experience
Endomyocardial biopsy in clinical practice: the diagnostic yield and insights from a 5-year single-center experience Open
With advances in cardiac imaging modalities, EMB is not routinely indicated for the diagnosis of cardiomyopathy. However, EMB is still an important tool for diagnosing specific cardiac diseases and could be crucial for confirming the diagn…
View article: Supplementary Figures 1-10 from Human and Mouse <i>VEGFA</i>-Amplified Hepatocellular Carcinomas Are Highly Sensitive to Sorafenib Treatment
Supplementary Figures 1-10 from Human and Mouse <i>VEGFA</i>-Amplified Hepatocellular Carcinomas Are Highly Sensitive to Sorafenib Treatment Open
PDF file 1687K, The supplementary data contains immunostains, mRNA and DNA qPCR, ELISA analyses and descriptive cartoons including a model highlighting the manuscript
View article: Supplementary Data from Molecular Mechanisms of Liver Carcinogenesis in the Mdr2-Knockout Mice
Supplementary Data from Molecular Mechanisms of Liver Carcinogenesis in the Mdr2-Knockout Mice Open
Supplementary Data from Molecular Mechanisms of Liver Carcinogenesis in the Mdr2-Knockout Mice
View article: Data from Molecular Mechanisms of Liver Carcinogenesis in the Mdr2-Knockout Mice
Data from Molecular Mechanisms of Liver Carcinogenesis in the Mdr2-Knockout Mice Open
Mouse models of hepatocellular carcinoma (HCC) simulate specific subgroups of human HCC. We investigated hepatocarcinogenesis in Mdr2-knockout (Mdr2-KO) mice, a model of inflammation-associated HCC, using gene expression profiling and immu…
View article: Supplementary Tables from Human and Mouse <i>VEGFA</i>-Amplified Hepatocellular Carcinomas Are Highly Sensitive to Sorafenib Treatment
Supplementary Tables from Human and Mouse <i>VEGFA</i>-Amplified Hepatocellular Carcinomas Are Highly Sensitive to Sorafenib Treatment Open
PDF file 145K, Tables containing information about genes residing on the amplicon and compiled data
View article: Data from Molecular Mechanisms of Liver Carcinogenesis in the Mdr2-Knockout Mice
Data from Molecular Mechanisms of Liver Carcinogenesis in the Mdr2-Knockout Mice Open
Mouse models of hepatocellular carcinoma (HCC) simulate specific subgroups of human HCC. We investigated hepatocarcinogenesis in Mdr2-knockout (Mdr2-KO) mice, a model of inflammation-associated HCC, using gene expression profiling and immu…
View article: Data from Human and Mouse <i>VEGFA</i>-Amplified Hepatocellular Carcinomas Are Highly Sensitive to Sorafenib Treatment
Data from Human and Mouse <i>VEGFA</i>-Amplified Hepatocellular Carcinomas Are Highly Sensitive to Sorafenib Treatment Open
Death rates from hepatocellular carcinoma (HCC) are steadily increasing, yet therapeutic options for advanced HCC are limited. We identify a subset of mouse and human HCCs harboring VEGFA genomic amplification, displaying distinct biologic…
View article: Supplementary Data from Molecular Mechanisms of Liver Carcinogenesis in the Mdr2-Knockout Mice
Supplementary Data from Molecular Mechanisms of Liver Carcinogenesis in the Mdr2-Knockout Mice Open
Supplementary Data from Molecular Mechanisms of Liver Carcinogenesis in the Mdr2-Knockout Mice
View article: Supplementary Tables from Human and Mouse <i>VEGFA</i>-Amplified Hepatocellular Carcinomas Are Highly Sensitive to Sorafenib Treatment
Supplementary Tables from Human and Mouse <i>VEGFA</i>-Amplified Hepatocellular Carcinomas Are Highly Sensitive to Sorafenib Treatment Open
PDF file 145K, Tables containing information about genes residing on the amplicon and compiled data
View article: Data from Human and Mouse <i>VEGFA</i>-Amplified Hepatocellular Carcinomas Are Highly Sensitive to Sorafenib Treatment
Data from Human and Mouse <i>VEGFA</i>-Amplified Hepatocellular Carcinomas Are Highly Sensitive to Sorafenib Treatment Open
Death rates from hepatocellular carcinoma (HCC) are steadily increasing, yet therapeutic options for advanced HCC are limited. We identify a subset of mouse and human HCCs harboring VEGFA genomic amplification, displaying distinct biologic…
View article: Supplementary Figures 1-10 from Human and Mouse <i>VEGFA</i>-Amplified Hepatocellular Carcinomas Are Highly Sensitive to Sorafenib Treatment
Supplementary Figures 1-10 from Human and Mouse <i>VEGFA</i>-Amplified Hepatocellular Carcinomas Are Highly Sensitive to Sorafenib Treatment Open
PDF file 1687K, The supplementary data contains immunostains, mRNA and DNA qPCR, ELISA analyses and descriptive cartoons including a model highlighting the manuscript
View article: Supplementary Materials and Methods from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis
Supplementary Materials and Methods from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis Open
Supplemental Experimental Materials and Methods
View article: Supplementary Materials and Methods from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis
Supplementary Materials and Methods from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis Open
Supplemental Experimental Materials and Methods
View article: Supplementary Data from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis
Supplementary Data from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis Open
Supplemental Figures and Tables
View article: Data from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis
Data from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis Open
Hepatocellular carcinoma (HCC) typically develops on a background of chronic hepatitis for which the proinflammatory cytokine IL6 is conventionally considered a crucial driving factor. Paradoxically, IL6 also acts as a hepatoprotective fac…
View article: Supplementary Data from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis
Supplementary Data from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis Open
Supplemental Figures and Tables
View article: Data from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis
Data from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis Open
Hepatocellular carcinoma (HCC) typically develops on a background of chronic hepatitis for which the proinflammatory cytokine IL6 is conventionally considered a crucial driving factor. Paradoxically, IL6 also acts as a hepatoprotective fac…
View article: Supplementary Figures 1-4 from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice
Supplementary Figures 1-4 from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice Open
Supplementary Figures 1-4 from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice
View article: Supplementary Tables 1-3 from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice
Supplementary Tables 1-3 from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice Open
Supplementary Tables 1-3 from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice
View article: Data from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice
Data from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice Open
Molecular events preceding the development of hepatocellular carcinoma were studied in the Mdr2-knockout (Mdr2-KO) mice. These mice lack the liver-specific P-glycoprotein responsible for phosphatidylcholine transport across the canalicular…
View article: Supplementary Methods from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice
Supplementary Methods from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice Open
Supplementary Methods from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice
View article: Supplementary Tables 1-3 from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice
Supplementary Tables 1-3 from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice Open
Supplementary Tables 1-3 from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice
View article: Data from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice
Data from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice Open
Molecular events preceding the development of hepatocellular carcinoma were studied in the Mdr2-knockout (Mdr2-KO) mice. These mice lack the liver-specific P-glycoprotein responsible for phosphatidylcholine transport across the canalicular…
View article: Supplementary Figures 1-4 from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice
Supplementary Figures 1-4 from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice Open
Supplementary Figures 1-4 from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice
View article: Supplementary Methods from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice
Supplementary Methods from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice Open
Supplementary Methods from Multiple Adaptive Mechanisms to Chronic Liver Disease Revealed at Early Stages of Liver Carcinogenesis in the Mdr2-Knockout Mice
View article: Extracellular Vesicular Transmission of miR-423-5p from HepG2 Cells Inhibits the Differentiation of Hepatic Stellate Cells
Extracellular Vesicular Transmission of miR-423-5p from HepG2 Cells Inhibits the Differentiation of Hepatic Stellate Cells Open
Liver fibrosis (LF) is a major cause of morbidity and mortality worldwide. Hepatic stellate cells (HSCs) are the primary source of extracellular matrix in the liver and their activation is a central event in LF development. Extracellular v…
View article: Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis
Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis Open
Hepatocellular carcinoma (HCC) typically develops on a background of chronic hepatitis for which the proinflammatory cytokine IL6 is conventionally considered a crucial driving factor. Paradoxically, IL6 also acts as a hepatoprotective fac…