Padmapriya Sekar
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View article: Autoantibodies inhibit Plasmodium falciparum growth and are associated with protection from clinical malaria
Autoantibodies inhibit Plasmodium falciparum growth and are associated with protection from clinical malaria Open
View article: NK cell-induced damage to P.falciparum-infected erythrocytes requires ligand-specific recognition and releases parasitophorous vacuoles that are phagocytosed by monocytes in the presence of immune IgG
NK cell-induced damage to P.falciparum-infected erythrocytes requires ligand-specific recognition and releases parasitophorous vacuoles that are phagocytosed by monocytes in the presence of immune IgG Open
Natural killer (NK) cells lyse virus-infected cells and transformed cells through polarized delivery of lytic effector molecules into target cells. We have shown that NK cells lyse Plasmodium falciparum -infected red blood cells (iRBC) via…
View article: NK cell-induced damage to<i>P.falciparum</i>-infected erythrocytes requires ligand-specific recognition and releases parasitophorous vacuoles that are phagocytosed by monocytes in the presence of immune IgG
NK cell-induced damage to<i>P.falciparum</i>-infected erythrocytes requires ligand-specific recognition and releases parasitophorous vacuoles that are phagocytosed by monocytes in the presence of immune IgG Open
Natural killer (NK) cells lyse virus-infected cells and transformed cells through polarized delivery of lytic effector molecules into target cells. We have shown that NK cells lyse Plasmodium falciparum -infected red blood cells (iRBC) via…
View article: Autoantibodies Inhibit&nbsp;Plasmodium falciparum&nbsp;Growth and Associate with Protection from Clinical Malaria
Autoantibodies Inhibit Plasmodium falciparum Growth and Associate with Protection from Clinical Malaria Open
View article: <i>Plasmodium falciparum</i> –specific IgM B cells dominate in children, expand with malaria, and produce functional IgM
<i>Plasmodium falciparum</i> –specific IgM B cells dominate in children, expand with malaria, and produce functional IgM Open
IgG antibodies play a role in malaria immunity, but whether and how IgM protects from malaria and the biology of Plasmodium falciparum (Pf)–specific IgM B cells is unclear. In a Mali cohort spanning infants to adults, we conducted longitud…
View article: <i>Plasmodium falciparum</i>-specific IgM B cells dominate in children, expand with malaria and produce parasite inhibitory IgM
<i>Plasmodium falciparum</i>-specific IgM B cells dominate in children, expand with malaria and produce parasite inhibitory IgM Open
IgG antibodies are known to play a central role in naturally acquired immunity to blood-stage malaria in humans, but little is known about the IgM response to blood-stage malaria, the mechanisms by which IgM may protect, or the underlying …
View article: The<i>Borrelia burgdorferi</i>CheY3 response regulator is essential for chemotaxis and completion of its natural infection cycle
The<i>Borrelia burgdorferi</i>CheY3 response regulator is essential for chemotaxis and completion of its natural infection cycle Open
Borrelia burgdorferi possesses a sophisticated and complex chemotaxis system, but how the organism utilizes this system in its natural enzootic life cycle is poorly understood. Of the three CheY chemotaxis response regulators in B. burgdor…
View article: Motor Rotation Is Essential for the Formation of the Periplasmic Flagellar Ribbon, Cellular Morphology, and Borrelia burgdorferi Persistence within Ixodes scapularis Tick and Murine Hosts
Motor Rotation Is Essential for the Formation of the Periplasmic Flagellar Ribbon, Cellular Morphology, and Borrelia burgdorferi Persistence within Ixodes scapularis Tick and Murine Hosts Open
Borrelia burgdorferi must migrate within and between its arthropod and mammalian hosts in order to complete its natural enzootic cycle. During tick feeding, the spirochete transmits from the tick to the host dermis, eventually colonizing a…