Quanbo Zhou
YOU?
Author Swipe
View article: Immunotherapy versus chemotherapy as adjuvant therapy for resected MSI-H/dMMR colorectal cancer: real-world evidence informing precision strategies
Immunotherapy versus chemotherapy as adjuvant therapy for resected MSI-H/dMMR colorectal cancer: real-world evidence informing precision strategies Open
Background Immunotherapy has demonstrated unique advantages in MSI-H/dMMR colorectal cancer (CRC) for its later-line, first-line in metastatic status, and neoadjuvant therapy. However, evidence regarding its value in postoperative adjuva…
View article: StCEL-based platform enables detection and sequencing of native sialoglycoRNA
StCEL-based platform enables detection and sequencing of native sialoglycoRNA Open
Glycosylated RNAs (glycoRNAs) are an emerging class of biomolecules characterized by glycans attached to RNA molecules. Currently, glycoRNA detection remains largely confined to cellular models, substantially impeding its functional charac…
View article: Angiogenesis and targeted therapy in the tumour microenvironment: From basic to clinical practice
Angiogenesis and targeted therapy in the tumour microenvironment: From basic to clinical practice Open
Angiogenesis, as a core marker of cancer survival and growth, is integral to the processes of tumour growth, invasion and metastasis. In recent years, targeted angiogenesis treatment strategies have gradually become an important direction …
View article: N4-acetylcytidine modification of ITGB5 mRNA mediated by NAT10 promotes perineural invasion in pancreatic ductal adenocarcinoma
N4-acetylcytidine modification of ITGB5 mRNA mediated by NAT10 promotes perineural invasion in pancreatic ductal adenocarcinoma Open
Background Perineural invasion (PNI) is a hallmark feature of pancreatic ductal adenocarcinoma (PDAC), which occurs at a high incidence and significantly contributes to PDAC lethality and poor survival. Despite its prevalence and associati…
View article: Core elements for a smooth transition from laparoscopic to robotic colorectal surgery
Core elements for a smooth transition from laparoscopic to robotic colorectal surgery Open
With the widespread adoption of da Vinci robotic system, the volume of robotic colorectal surgeries has witnessed an explosive increase. Studies have shown that robotic surgery outperforms traditional laparoscopy in terms of tumor resectio…
View article: Predictive model of the surgical difficulty of robot-assisted total mesorectal excision for rectal cancer: a multicenter, retrospective study
Predictive model of the surgical difficulty of robot-assisted total mesorectal excision for rectal cancer: a multicenter, retrospective study Open
View article: Supplementary Table S1 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression
Supplementary Table S1 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression Open
Supplementary Table S1
View article: Supplementary Table S5 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression
Supplementary Table S5 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression Open
Supplementary Table S5
View article: Supplementary Table S7 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression
Supplementary Table S7 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression Open
Supplementary Table S7
View article: Figure S2 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression
Figure S2 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression Open
Figure S2 and it's legend
View article: Supplementary Table S3 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression
Supplementary Table S3 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression Open
Supplementary Table S3
View article: Supplementary Table S6 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression
Supplementary Table S6 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression Open
Supplementary Table S6
View article: Figure S3 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression
Figure S3 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression Open
Figure S3 and it's legend
View article: Figure S4 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression
Figure S4 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression Open
Figure S4 and it's legend
View article: Supplementary Table S9 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression
Supplementary Table S9 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression Open
Supplementary Table S9
View article: Data from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression
Data from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression Open
N6-Methyladenosine (m6A) is the most prevalent RNA modification and is associated with various biological processes. Proteins that function as readers and writers of m6A modifications have been shown…
View article: Supplementary Table S10 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression
Supplementary Table S10 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression Open
Supplementary Table S10
View article: Figure S1 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression
Figure S1 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression Open
Figure S1 and it's legend
View article: Supplementary Table S2 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression
Supplementary Table S2 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression Open
Supplementary Table S2
View article: Figure S5 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression
Figure S5 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression Open
Figure S5 and it's legend
View article: Supplementary Table S4 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression
Supplementary Table S4 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression Open
Supplementary Table S4
View article: Figure S6 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression
Figure S6 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression Open
Figure S6 and it's legend
View article: Supplementary Table S8 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression
Supplementary Table S8 from KHSRP Stabilizes m<sup>6</sup>A-Modified Transcripts to Activate FAK Signaling and Promote Pancreatic Ductal Adenocarcinoma Progression Open
Supplementary Table S8
View article: Predictive model of the surgical difficulty of robot-assisted total mesorectal excision for rectal cancer: A multicenter, retrospective study
Predictive model of the surgical difficulty of robot-assisted total mesorectal excision for rectal cancer: A multicenter, retrospective study Open
Background Rectal cancer robotic surgery is becoming more and more common, but evidence for predicting surgical difficulty is scarce. Our goal was to look at the elements that influence the complexity of robot-assisted total mesorectal exc…
View article: Pre-metastatic niche: formation, characteristics and therapeutic implication
Pre-metastatic niche: formation, characteristics and therapeutic implication Open
Distant metastasis is a primary cause of mortality and contributes to poor surgical outcomes in cancer patients. Before the development of organ-specific metastasis, the formation of a pre-metastatic niche is pivotal in promoting the sprea…
View article: Data from N6-methyladenosine modification of FZR1 mRNA promotes gemcitabine resistance in pancreatic cancer
Data from N6-methyladenosine modification of FZR1 mRNA promotes gemcitabine resistance in pancreatic cancer Open
The therapeutic options for treating pancreatic ductal adenocarcinoma (PDAC) are limited, and resistance to gemcitabine, a cornerstone of PDAC chemotherapy regimens, remains a major challenge. N6-methyladenosine (m6A) is a prevalent modifi…
View article: Supplementary Data from N6-methyladenosine modification of FZR1 mRNA promotes gemcitabine resistance in pancreatic cancer
Supplementary Data from N6-methyladenosine modification of FZR1 mRNA promotes gemcitabine resistance in pancreatic cancer Open
Supplementary Figures 1-6 and supplementary Tables 1-7.
View article: Supplementary Data from N6-methyladenosine modification of FZR1 mRNA promotes gemcitabine resistance in pancreatic cancer
Supplementary Data from N6-methyladenosine modification of FZR1 mRNA promotes gemcitabine resistance in pancreatic cancer Open
Supplementary Figures 1-6 and supplementary Tables 1-7.
View article: Supplementary Data from N6-methyladenosine modification of FZR1 mRNA promotes gemcitabine resistance in pancreatic cancer
Supplementary Data from N6-methyladenosine modification of FZR1 mRNA promotes gemcitabine resistance in pancreatic cancer Open
Proteins identified in RNA pulldown and mass spectrometry analysis with FZR1[m6A] or FZR1[A].
View article: Supplementary Data from N6-methyladenosine modification of FZR1 mRNA promotes gemcitabine resistance in pancreatic cancer
Supplementary Data from N6-methyladenosine modification of FZR1 mRNA promotes gemcitabine resistance in pancreatic cancer Open
Proteins identified in RNA pulldown and mass spectrometry analysis with FZR1[m6A] or FZR1[A].