Raka Bhattacharya
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View article: Supplementary Data from Upregulation of <i>MMP-2</i> by HMGA1 Promotes Transformation in Undifferentiated, Large-Cell Lung Cancer
Supplementary Data from Upregulation of <i>MMP-2</i> by HMGA1 Promotes Transformation in Undifferentiated, Large-Cell Lung Cancer Open
Supplementary Data from Upregulation of MMP-2 by HMGA1 Promotes Transformation in Undifferentiated, Large-Cell Lung Cancer
View article: Data from HMGA2 Participates in Transformation in Human Lung Cancer
Data from HMGA2 Participates in Transformation in Human Lung Cancer Open
Although previous studies have established a prominent role for HMGA1 (formerly HMG-I/Y) in aggressive human cancers, the role of HMGA2 (formerly HMGI-C) in malignant transformation has not been clearly defined.…
View article: Data from HMGA2 Participates in Transformation in Human Lung Cancer
Data from HMGA2 Participates in Transformation in Human Lung Cancer Open
Although previous studies have established a prominent role for HMGA1 (formerly HMG-I/Y) in aggressive human cancers, the role of HMGA2 (formerly HMGI-C) in malignant transformation has not been clearly defined.…
View article: Data from Upregulation of <i>MMP-2</i> by HMGA1 Promotes Transformation in Undifferentiated, Large-Cell Lung Cancer
Data from Upregulation of <i>MMP-2</i> by HMGA1 Promotes Transformation in Undifferentiated, Large-Cell Lung Cancer Open
Although lung cancer is the leading cause of cancer death worldwide, the precise molecular mechanisms that give rise to lung cancer are incompletely understood. Here, we show that HMGA1 is an important oncogene that drives transform…
View article: Supplementary Data from HMGA2 Participates in Transformation in Human Lung Cancer
Supplementary Data from HMGA2 Participates in Transformation in Human Lung Cancer Open
Supplementary Data from HMGA2 Participates in Transformation in Human Lung Cancer
View article: Supplementary Data from Upregulation of <i>MMP-2</i> by HMGA1 Promotes Transformation in Undifferentiated, Large-Cell Lung Cancer
Supplementary Data from Upregulation of <i>MMP-2</i> by HMGA1 Promotes Transformation in Undifferentiated, Large-Cell Lung Cancer Open
Supplementary Data from Upregulation of MMP-2 by HMGA1 Promotes Transformation in Undifferentiated, Large-Cell Lung Cancer
View article: Supplementary Data from HMGA2 Participates in Transformation in Human Lung Cancer
Supplementary Data from HMGA2 Participates in Transformation in Human Lung Cancer Open
Supplementary Data from HMGA2 Participates in Transformation in Human Lung Cancer
View article: Data from Upregulation of <i>MMP-2</i> by HMGA1 Promotes Transformation in Undifferentiated, Large-Cell Lung Cancer
Data from Upregulation of <i>MMP-2</i> by HMGA1 Promotes Transformation in Undifferentiated, Large-Cell Lung Cancer Open
Although lung cancer is the leading cause of cancer death worldwide, the precise molecular mechanisms that give rise to lung cancer are incompletely understood. Here, we show that HMGA1 is an important oncogene that drives transform…
View article: Supplementary Table 1 from The High-Mobility Group A1a/Signal Transducer and Activator of Transcription-3 Axis: An Achilles Heel for Hematopoietic Malignancies?
Supplementary Table 1 from The High-Mobility Group A1a/Signal Transducer and Activator of Transcription-3 Axis: An Achilles Heel for Hematopoietic Malignancies? Open
Supplementary Table 1 from The High-Mobility Group A1a/Signal Transducer and Activator of Transcription-3 Axis: An Achilles Heel for Hematopoietic Malignancies?
View article: Data from The High-Mobility Group A1a/Signal Transducer and Activator of Transcription-3 Axis: An Achilles Heel for Hematopoietic Malignancies?
Data from The High-Mobility Group A1a/Signal Transducer and Activator of Transcription-3 Axis: An Achilles Heel for Hematopoietic Malignancies? Open
Although HMGA1 (high-mobility group A1; formerly HMG-I/Y) is an oncogene that is widely overexpressed in aggressive cancers, the molecular mechanisms underlying transformation by HMGA1 are only beginning to emer…
View article: Supplementary Table 2 from The High-Mobility Group A1a/Signal Transducer and Activator of Transcription-3 Axis: An Achilles Heel for Hematopoietic Malignancies?
Supplementary Table 2 from The High-Mobility Group A1a/Signal Transducer and Activator of Transcription-3 Axis: An Achilles Heel for Hematopoietic Malignancies? Open
Supplementary Table 2 from The High-Mobility Group A1a/Signal Transducer and Activator of Transcription-3 Axis: An Achilles Heel for Hematopoietic Malignancies?
View article: Supplementary Table 2 from The High-Mobility Group A1a/Signal Transducer and Activator of Transcription-3 Axis: An Achilles Heel for Hematopoietic Malignancies?
Supplementary Table 2 from The High-Mobility Group A1a/Signal Transducer and Activator of Transcription-3 Axis: An Achilles Heel for Hematopoietic Malignancies? Open
Supplementary Table 2 from The High-Mobility Group A1a/Signal Transducer and Activator of Transcription-3 Axis: An Achilles Heel for Hematopoietic Malignancies?
View article: Supplementary Table 1 from The High-Mobility Group A1a/Signal Transducer and Activator of Transcription-3 Axis: An Achilles Heel for Hematopoietic Malignancies?
Supplementary Table 1 from The High-Mobility Group A1a/Signal Transducer and Activator of Transcription-3 Axis: An Achilles Heel for Hematopoietic Malignancies? Open
Supplementary Table 1 from The High-Mobility Group A1a/Signal Transducer and Activator of Transcription-3 Axis: An Achilles Heel for Hematopoietic Malignancies?
View article: Data from The High-Mobility Group A1a/Signal Transducer and Activator of Transcription-3 Axis: An Achilles Heel for Hematopoietic Malignancies?
Data from The High-Mobility Group A1a/Signal Transducer and Activator of Transcription-3 Axis: An Achilles Heel for Hematopoietic Malignancies? Open
Although HMGA1 (high-mobility group A1; formerly HMG-I/Y) is an oncogene that is widely overexpressed in aggressive cancers, the molecular mechanisms underlying transformation by HMGA1 are only beginning to emer…
View article: Vaccine-Induced Intratumoral Lymphoid Aggregates Correlate with Survival Following Treatment with a Neoadjuvant and Adjuvant Vaccine in Patients with Resectable Pancreatic Adenocarcinoma
Vaccine-Induced Intratumoral Lymphoid Aggregates Correlate with Survival Following Treatment with a Neoadjuvant and Adjuvant Vaccine in Patients with Resectable Pancreatic Adenocarcinoma Open
Purpose: Immunotherapy is currently ineffective for nearly all pancreatic ductal adenocarcinomas (PDAC), largely due to its tumor microenvironment (TME) that lacks antigen-experienced T effector cells (Teff). Vaccine-based immunotherapies …