Ruby Oberin
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Fetal growth delay caused by loss of non-canonical imprinting is resolved late in pregnancy and culminates in offspring overgrowth Open
Germline epigenetic programming, including genomic imprinting, substantially influences offspring development. Polycomb Repressive Complex 2 (PRC2) plays an important role in Histone 3 Lysine 27 trimethylation (H3K27me3)-dependent imprinti…
Additional file 2 of Transient Polycomb activity represses developmental genes in growing oocytes Open
Additional file 2: Excel file containing a Summary of all Supplementary tables and sheets containing individual Supplementary Tables.
Transient Polycomb activity represses developmental genes in growing oocytes Open
Background Non-genetic disease inheritance and offspring phenotype is substantially influenced by germline epigenetic programming, including genomic imprinting. Loss of Polycomb Repressive Complex 2 (PRC2) function in oocytes causes non-ge…
Loss of EED in the oocyte causes initial fetal growth restriction followed by placental hyperplasia and offspring overgrowth Open
Germline epigenetic programming, including genomic imprinting, substantially influences offspring development. Polycomb Repressive Complex 2 (PRC2) plays an important role in Histone 3 Lysine 27 trimethylation (H3K27me3)-dependent imprinti…