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View article: First Measurement of the Quadrupole Moment of the <mml:math xmlns:mml="http://www.w3.org/1998/Math/MathML" display="inline"> <mml:msubsup> <mml:mn>2</mml:mn> <mml:mn>1</mml:mn> <mml:mo>+</mml:mo> </mml:msubsup> </mml:math> State in <mml:math xmlns:mml="http://www.w3.org/1998/Math/MathML" display="inline"> <mml:mrow> <mml:mmultiscripts> <mml:mrow> <mml:mi>Sn</mml:mi> </mml:mrow> <mml:mprescripts/> <mml:none/> <mml:mrow> <mml:mn>110</mml:mn> </mml:mrow> </mml:mmultiscripts> </mml:mrow> </mml:math>
First Measurement of the Quadrupole Moment of the State in Open
The Sn isotopic chain, exhibiting double shell closures at Sn 100 and Sn 132 , is a key testing ground for theoretical models of the atomic nucleus. It was originally predicted that the transitional matrix elements …
View article: Hybrid Deep Learning Architecture for Efficient Human Activity Recognition: A CNN-Attention-BiLSTM Framework
Hybrid Deep Learning Architecture for Efficient Human Activity Recognition: A CNN-Attention-BiLSTM Framework Open
Human Activity Recognition (HAR) has emerged as a critical research area in the domains of Artificial Intelligence (AI), Machine Learning (ML), and Deep Learning (DL) due to its extensive applications across various domains. The developmen…
View article: Chiral Self‐Assembly of a Pyrene‐Appended Glutamylalanine Dipeptide and Its Charge Transfer Complex: Fabrication of Magneto‐Responsive Hydrogels and Human Cell Imaging
Chiral Self‐Assembly of a Pyrene‐Appended Glutamylalanine Dipeptide and Its Charge Transfer Complex: Fabrication of Magneto‐Responsive Hydrogels and Human Cell Imaging Open
The formation of a robust, self‐healing hydrogel of a novel pyrene‐appended dipeptide, Py‐E‐A (L‐Glutamic acid short as E; L‐Alanine short as A) is demonstrated. Detailed studies suggest that nanoscopic fibers with a length of several micr…
View article: Assessment of the Bangladeshi antibiotic market: Implications of the WHO AWaRe classification and dosage form availability on antimicrobial resistance
Assessment of the Bangladeshi antibiotic market: Implications of the WHO AWaRe classification and dosage form availability on antimicrobial resistance Open
The widespread availability of Watch class antibiotics, particularly in oral and child-appropriate formulations suggest a need for stricter regulation and public health interventions to curb self-medication, inappropriate marketing and use…
View article: A Clinical Evaluation of Platelet-Rich Plasma Therapy in Treatment of Frozen Shoulder
A Clinical Evaluation of Platelet-Rich Plasma Therapy in Treatment of Frozen Shoulder Open
Background: Frozen shoulder is a common reason for shoulder pain and impairment. Several treatments are used to relieve pain and enhance range of motion (ROM) in patients. Recent study has focused on the injection of platelet-rich plasma (…
View article: The Prostate Cancer Detection Using K-Nearest Neighbor (KNN)
The Prostate Cancer Detection Using K-Nearest Neighbor (KNN) Open
The ongoing threat that cancer poses to the health and prosperity of the world highlights the critical need for early identification and efficient treatment. Machine learning and artificial intelligence have become effective tools for the …
View article: The Melanoma Skin Cancer Detection Using Convolutional Neural Network
The Melanoma Skin Cancer Detection Using Convolutional Neural Network Open
The most common cause of death for persons in the modern world is melanoma skin cancer. Melanoma is an aggressive kind of skin cancer that typically develops on parts of the body exposed to sunlight, UV radiation, dust, pollution, and micr…
View article: Real Time Vehicle Identification: A Synchronous-Transmission Based Approach
Real Time Vehicle Identification: A Synchronous-Transmission Based Approach Open
Identification of the vehicles passing over the roads is a very important component of traffic monitoring/surveillance. There have been many attempts to design and develop efficient strategies to carry out the job. However, from the point …
View article: Supplementary Figure 4 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics
Supplementary Figure 4 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics Open
Figure S4. Gβ5 knockdown obviates mobilization of antioxidant defense systems in VCM exposed to chemotherapeutic drugs.
View article: Supplementary Figure 3 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics
Supplementary Figure 3 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics Open
Figure S3. Gβ5 promotes chemotherapeutic drug-induced oxidative stress in isolated VCM.
View article: Supplementary Figure 8 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics
Supplementary Figure 8 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics Open
Figure S8. Gβ5 promotes pathological myocyte-fibroblast crosstalk activated upon chemotherapeutic drug exposure.
View article: Supplementary Figure 2 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics
Supplementary Figure 2 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics Open
Figure S2. Validation of Gβ5-targeted shRNA constructs.
View article: Supplementary Figure 6 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics
Supplementary Figure 6 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics Open
Figure S6. Gβ5 functions in a pathway with ROS, ATM and CAMKII to promote chemotherapeutic drug-induced cardiomyocyte apoptosis.
View article: Data from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics
Data from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics Open
The clinical use of multiple classes of cancer chemotherapeutics is limited by irreversible, dose-dependent, and sometimes life-threatening cardiotoxicity. Though distinct in their mechanisms of action, doxorubicin, paclitaxel, and 5-FU al…
View article: Supplementary Figure 5 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics
Supplementary Figure 5 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics Open
Figure S5. Nox-dependent ROS generation promotes activation of CaMKII and ATM following chemotherapy exposure.
View article: Supplementary Figure 7 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics
Supplementary Figure 7 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics Open
Figure S7. p53 knockdown in VCM fails to impact cardiac fibrosis following doxorubicin exposure.
View article: Supplementary Figure 1 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics
Supplementary Figure 1 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics Open
Figure S1. Gβ5 upregulation following chemotherapeutic drug exposure requires new protein synthesis.
View article: Supplementary Figure 4 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics
Supplementary Figure 4 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics Open
Figure S4. Gβ5 knockdown obviates mobilization of antioxidant defense systems in VCM exposed to chemotherapeutic drugs.
View article: Supplementary Figure 5 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics
Supplementary Figure 5 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics Open
Figure S5. Nox-dependent ROS generation promotes activation of CaMKII and ATM following chemotherapy exposure.
View article: Supplementary Figure 7 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics
Supplementary Figure 7 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics Open
Figure S7. p53 knockdown in VCM fails to impact cardiac fibrosis following doxorubicin exposure.
View article: Supplementary Figure 3 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics
Supplementary Figure 3 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics Open
Figure S3. Gβ5 promotes chemotherapeutic drug-induced oxidative stress in isolated VCM.
View article: Supplementary Figure 2 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics
Supplementary Figure 2 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics Open
Figure S2. Validation of Gβ5-targeted shRNA constructs.
View article: Supplementary Figure 1 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics
Supplementary Figure 1 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics Open
Figure S1. Gβ5 upregulation following chemotherapeutic drug exposure requires new protein synthesis.
View article: Supplementary Figure 8 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics
Supplementary Figure 8 from Atypical G Protein β5 Promotes Cardiac Oxidative Stress, Apoptosis, and Fibrotic Remodeling in Response to Multiple Cancer Chemotherapeutics Open
Figure S8. Gβ5 promotes pathological myocyte-fibroblast crosstalk activated upon chemotherapeutic drug exposure.