Shingo Yoshizaki
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View article: Zinc deficiency impairs axonal regeneration and functional recovery after spinal cord injury by modulating macrophage polarization via NF-κB pathway
Zinc deficiency impairs axonal regeneration and functional recovery after spinal cord injury by modulating macrophage polarization via NF-κB pathway Open
Background Spinal cord injury (SCI) is a devastating disease that results in permanent paralysis. Currently, there is no effective treatment for SCI, and it is important to identify factors that can provide therapeutic intervention during …
View article: Zinc deficiency impairs functional recovery after spinal cord injury by promoting NF-κB activity in macrophages
Zinc deficiency impairs functional recovery after spinal cord injury by promoting NF-κB activity in macrophages Open
Background Spinal cord injury (SCI) is a devastating disease that results in permanent paralysis. Currently, there is no effective treatment for SCI, and it is important to identify factors that can provide therapeutic intervention during …
View article: Glial scar survives until the chronic phase by recruiting scar-forming astrocytes after spinal cord injury
Glial scar survives until the chronic phase by recruiting scar-forming astrocytes after spinal cord injury Open
View article: The Posterolaterally Oriented and Laterally Downward Sloping Facet Joint Is a Risk Factor for Degenerative Cervical Spondylolisthesis and Myelopathy
The Posterolaterally Oriented and Laterally Downward Sloping Facet Joint Is a Risk Factor for Degenerative Cervical Spondylolisthesis and Myelopathy Open
The posterolaterally oriented and laterally downward sloping facet at C3/4 and C4/5 levels may be a risk factor for the development of cervical spondylolisthesis as well as symptomatic myelopathy.
View article: Microglial inflammation after chronic spinal cord injury is enhanced by reactive astrocytes via the fibronectin/β1 integrin pathway
Microglial inflammation after chronic spinal cord injury is enhanced by reactive astrocytes via the fibronectin/β1 integrin pathway Open
View article: Tranexamic acid reduces heme cytotoxicity via the TLR4/TNF axis and ameliorates functional recovery after spinal cord injury
Tranexamic acid reduces heme cytotoxicity via the TLR4/TNF axis and ameliorates functional recovery after spinal cord injury Open
View article: Macrophage centripetal migration drives spontaneous healing process after spinal cord injury
Macrophage centripetal migration drives spontaneous healing process after spinal cord injury Open
Migration of macrophages via IRF8 toward a core lesion is important for the spontaneous recovery after spinal cord injury.
View article: The acute phase serum zinc concentration is a reliable biomarker for predicting the functional outcome after spinal cord injury
The acute phase serum zinc concentration is a reliable biomarker for predicting the functional outcome after spinal cord injury Open
The acute-phase serum zinc concentration could be a useful biomarker for predicting the functional prognosis. This simple method will allow for more objective clinical trials and the development of patient-tailored treatment for SCI.
View article: Additional file 3: of Tranexamic acid reduces heme cytotoxicity via the TLR4/TNF axis and ameliorates functional recovery after spinal cord injury
Additional file 3: of Tranexamic acid reduces heme cytotoxicity via the TLR4/TNF axis and ameliorates functional recovery after spinal cord injury Open
Table S1. The primer sequences used for qRT-PCR in the present study. Accession numbers are for the NCBI Gene database: https://www.ncbi.nlm.nih.gov/gene (XLSX 10 kb)
View article: The Acute Phase Serum Zinc Concentration Is a Reliable Biomarker for Predicting the Functional Outcome After Spinal Cord Injury
The Acute Phase Serum Zinc Concentration Is a Reliable Biomarker for Predicting the Functional Outcome After Spinal Cord Injury Open
View article: Macrophage Infiltration Is a Causative Factor for Ligamentum Flavum Hypertrophy through the Activation of Collagen Production in Fibroblasts
Macrophage Infiltration Is a Causative Factor for Ligamentum Flavum Hypertrophy through the Activation of Collagen Production in Fibroblasts Open