Shuling Ren
YOU?
Author Swipe
View article: Data from Cannabinoids Promote Progression of HPV-Positive Head and Neck Squamous Cell Carcinoma via p38 MAPK Activation
Data from Cannabinoids Promote Progression of HPV-Positive Head and Neck Squamous Cell Carcinoma via p38 MAPK Activation Open
Purpose:Human papillomavirus (HPV)-related head and neck squamous cell carcinoma (HNSCC) is associated with daily marijuana use and is also increasing in parallel with increased marijuana use in the United States. Our study is designed to …
View article: Figure S1-9 from Cannabinoids Promote Progression of HPV-Positive Head and Neck Squamous Cell Carcinoma via p38 MAPK Activation
Figure S1-9 from Cannabinoids Promote Progression of HPV-Positive Head and Neck Squamous Cell Carcinoma via p38 MAPK Activation Open
Figure S1: CNR1 and CNR2 affect the proliferation of HPV positive HNSCC cells. Figure S2: High concentrations of cannabinoids inhibit the growth of HPV positive HNSCC cells. Figure S3: Inhibition of CNR1 and CNR2 attenuates the effect of C…
View article: Table S2 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant
Table S2 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant Open
Full list of 580 validated ASEs associated with HPV negative HNSCC
View article: Table S4 from Extrachromosomal DNA in HPV-Mediated Oropharyngeal Cancer Drives Diverse Oncogene Transcription
Table S4 from Extrachromosomal DNA in HPV-Mediated Oropharyngeal Cancer Drives Diverse Oncogene Transcription Open
Survival analysis of Institutional HPV positive tumors by ecDNA and hybrid classification.
View article: Figure S4 from Extrachromosomal DNA in HPV-Mediated Oropharyngeal Cancer Drives Diverse Oncogene Transcription
Figure S4 from Extrachromosomal DNA in HPV-Mediated Oropharyngeal Cancer Drives Diverse Oncogene Transcription Open
Principal component analyses of utilization of canonical HPV splice sites in institutional and TCGA cohorts.
View article: Table S6 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant
Table S6 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant Open
Correlations between the expression of DOCK5 variant and clinicopathological parameters in TCGA HPV negative HNSCC data
View article: Data from The <i>NOTCH4</i>–<i>HEY1</i> Pathway Induces Epithelial–Mesenchymal Transition in Head and Neck Squamous Cell Carcinoma
Data from The <i>NOTCH4</i>–<i>HEY1</i> Pathway Induces Epithelial–Mesenchymal Transition in Head and Neck Squamous Cell Carcinoma Open
Purpose: Recently, several comprehensive genomic analyses demonstrated NOTCH1 and NOTCH3 mutations in head and neck squamous cell carcinoma (HNSCC) in approximately 20% of cases. Similar to other types of cancers, these studies also indica…
View article: Table S5 from Cannabinoids Promote Progression of HPV-Positive Head and Neck Squamous Cell Carcinoma via p38 MAPK Activation
Table S5 from Cannabinoids Promote Progression of HPV-Positive Head and Neck Squamous Cell Carcinoma via p38 MAPK Activation Open
List of relatively enriched pathways corresponding to each oncogenic state forming the Onco-GPS map.
View article: Figure S6 from The <i>NOTCH4</i>–<i>HEY1</i> Pathway Induces Epithelial–Mesenchymal Transition in Head and Neck Squamous Cell Carcinoma
Figure S6 from The <i>NOTCH4</i>–<i>HEY1</i> Pathway Induces Epithelial–Mesenchymal Transition in Head and Neck Squamous Cell Carcinoma Open
Figure S6. Comparison of overall survival between NOTCH4, HEY1 high and low groups using the TCGA data set.
View article: Table S1 from The <i>NOTCH4</i>–<i>HEY1</i> Pathway Induces Epithelial–Mesenchymal Transition in Head and Neck Squamous Cell Carcinoma
Table S1 from The <i>NOTCH4</i>–<i>HEY1</i> Pathway Induces Epithelial–Mesenchymal Transition in Head and Neck Squamous Cell Carcinoma Open
Table S1. HNSCC NOTCH mutation samples in TCGA
View article: Table S4 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant
Table S4 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant Open
Correlations between number of ASEs and clinicopathological parameters in TCGA HPV negative HNSCC data
View article: Data from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant
Data from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant Open
Purpose: Head and neck squamous cell carcinoma (HNSCC) is one of the most common cancers worldwide, and alternative splicing is considered to play important roles in tumor progression. Our study is designed to identify alternative splicing…
View article: Table S5 from Cannabinoids Promote Progression of HPV-Positive Head and Neck Squamous Cell Carcinoma via p38 MAPK Activation
Table S5 from Cannabinoids Promote Progression of HPV-Positive Head and Neck Squamous Cell Carcinoma via p38 MAPK Activation Open
List of relatively enriched pathways corresponding to each oncogenic state forming the Onco-GPS map.
View article: Table S8 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant
Table S8 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant Open
List of dysregulated pathways between patients with higher and lower expression of DOCK5 variant in TCGA HPV negative HNSCC data
View article: Figure S3 from The <i>NOTCH4</i>–<i>HEY1</i> Pathway Induces Epithelial–Mesenchymal Transition in Head and Neck Squamous Cell Carcinoma
Figure S3 from The <i>NOTCH4</i>–<i>HEY1</i> Pathway Induces Epithelial–Mesenchymal Transition in Head and Neck Squamous Cell Carcinoma Open
Figure S3. Sphere colony shapes. HEY1 expression in si-HEY1 cells and NOTCH4 expressions in sh-NOTCH4 cells
View article: Table S4 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant
Table S4 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant Open
Correlations between number of ASEs and clinicopathological parameters in TCGA HPV negative HNSCC data
View article: Table S4 from Cannabinoids Promote Progression of HPV-Positive Head and Neck Squamous Cell Carcinoma via p38 MAPK Activation
Table S4 from Cannabinoids Promote Progression of HPV-Positive Head and Neck Squamous Cell Carcinoma via p38 MAPK Activation Open
List of relatively enriched pathways for each transcriptional component forming the Onco-GPS map.
View article: Data from The <i>NOTCH4</i>–<i>HEY1</i> Pathway Induces Epithelial–Mesenchymal Transition in Head and Neck Squamous Cell Carcinoma
Data from The <i>NOTCH4</i>–<i>HEY1</i> Pathway Induces Epithelial–Mesenchymal Transition in Head and Neck Squamous Cell Carcinoma Open
Purpose: Recently, several comprehensive genomic analyses demonstrated NOTCH1 and NOTCH3 mutations in head and neck squamous cell carcinoma (HNSCC) in approximately 20% of cases. Similar to other types of cancers, these studies also indica…
View article: Data from Cannabinoids Promote Progression of HPV-Positive Head and Neck Squamous Cell Carcinoma via p38 MAPK Activation
Data from Cannabinoids Promote Progression of HPV-Positive Head and Neck Squamous Cell Carcinoma via p38 MAPK Activation Open
Purpose:Human papillomavirus (HPV)-related head and neck squamous cell carcinoma (HNSCC) is associated with daily marijuana use and is also increasing in parallel with increased marijuana use in the United States. Our study is designed to …
View article: Figure S3 from Extrachromosomal DNA in HPV-Mediated Oropharyngeal Cancer Drives Diverse Oncogene Transcription
Figure S3 from Extrachromosomal DNA in HPV-Mediated Oropharyngeal Cancer Drives Diverse Oncogene Transcription Open
Downstream expression at site of integration of human/viral hybrid RNA sequence.
View article: Table S5 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant
Table S5 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant Open
List of junctions in both HPV negative and positive HNSCC
View article: Table S7 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant
Table S7 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant Open
List of differentially expressed genes between patients with higher and lower expression of DOCK5 variant in TCGA HPV negative HNSCC data
View article: Table S1 from Cannabinoids Promote Progression of HPV-Positive Head and Neck Squamous Cell Carcinoma via p38 MAPK Activation
Table S1 from Cannabinoids Promote Progression of HPV-Positive Head and Neck Squamous Cell Carcinoma via p38 MAPK Activation Open
Expression of cannabinoids and their metabolites in plasma samples of HPV positive HNSCC patients identified by LC-MS/MS.
View article: Figure S6 from The <i>NOTCH4</i>–<i>HEY1</i> Pathway Induces Epithelial–Mesenchymal Transition in Head and Neck Squamous Cell Carcinoma
Figure S6 from The <i>NOTCH4</i>–<i>HEY1</i> Pathway Induces Epithelial–Mesenchymal Transition in Head and Neck Squamous Cell Carcinoma Open
Figure S6. Comparison of overall survival between NOTCH4, HEY1 high and low groups using the TCGA data set.
View article: Table S3 from Cannabinoids Promote Progression of HPV-Positive Head and Neck Squamous Cell Carcinoma via p38 MAPK Activation
Table S3 from Cannabinoids Promote Progression of HPV-Positive Head and Neck Squamous Cell Carcinoma via p38 MAPK Activation Open
List of dysregulated pathways in patients with cannabinoid metabolites compared with patients without cannabinoid metabolites.
View article: Table S3 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant
Table S3 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant Open
Distribution of 580 validated junctions in each tumor sample
View article: Supplementary Legend from Extrachromosomal DNA in HPV-Mediated Oropharyngeal Cancer Drives Diverse Oncogene Transcription
Supplementary Legend from Extrachromosomal DNA in HPV-Mediated Oropharyngeal Cancer Drives Diverse Oncogene Transcription Open
Supplementary Legend
View article: Table S7 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant
Table S7 from Characterization of Alternative Splicing Events in HPV-Negative Head and Neck Squamous Cell Carcinoma Identifies an Oncogenic DOCK5 Variant Open
List of differentially expressed genes between patients with higher and lower expression of DOCK5 variant in TCGA HPV negative HNSCC data
View article: Table S4 from Extrachromosomal DNA in HPV-Mediated Oropharyngeal Cancer Drives Diverse Oncogene Transcription
Table S4 from Extrachromosomal DNA in HPV-Mediated Oropharyngeal Cancer Drives Diverse Oncogene Transcription Open
Survival analysis of Institutional HPV positive tumors by ecDNA and hybrid classification.