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View article: Supplementary Figure 2 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells
Supplementary Figure 2 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells Open
PDF file - 32K, Effects of different compound AXIN stabilizers on TNKS1 and TNKS2.
View article: Supplementary Figure 6 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells
Supplementary Figure 6 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells Open
PDF file - 112K, Average body weight graph.
View article: Supplementary Figure Legend from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells
Supplementary Figure Legend from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells Open
PDF file - 81K
View article: Data from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells
Data from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells Open
Glioblastoma multiforme (GBM) is the most common and prognostically unfavorable form of brain tumor. The aggressive and highly invasive phenotype of these tumors makes them among the most anatomically damaging human cancers with a median s…
View article: Supplementary Figure Legend from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells
Supplementary Figure Legend from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells Open
PDF file - 81K
View article: Supplementary Figure 1 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells
Supplementary Figure 1 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells Open
PDF file - 94K, Consequences of inhibition of WNT signaling in glioblastoma cells: a less proliferative and less tumorigenic phenotype.
View article: Supplementary Figure 4 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells
Supplementary Figure 4 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells Open
PDF file - 10K, SEN461 affects canonical WNT ligands mediated transcription.
View article: Supplementary Figure 2 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells
Supplementary Figure 2 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells Open
PDF file - 32K, Effects of different compound AXIN stabilizers on TNKS1 and TNKS2.
View article: Supplementary Figure 3 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells
Supplementary Figure 3 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells Open
PDF file - 20K, Comparative WNT transcriptional activity and growth inhibition with different compound AXIN stabilizers.
View article: Supplementary Figure 5 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells
Supplementary Figure 5 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells Open
PDF file - 98K, In vitro response of T98G cells to WNT signaling inhibition.
View article: Data from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells
Data from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells Open
Glioblastoma multiforme (GBM) is the most common and prognostically unfavorable form of brain tumor. The aggressive and highly invasive phenotype of these tumors makes them among the most anatomically damaging human cancers with a median s…
View article: Supplementary Figure 1 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells
Supplementary Figure 1 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells Open
PDF file - 94K, Consequences of inhibition of WNT signaling in glioblastoma cells: a less proliferative and less tumorigenic phenotype.
View article: Supplementary Figure 4 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells
Supplementary Figure 4 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells Open
PDF file - 10K, SEN461 affects canonical WNT ligands mediated transcription.
View article: Supplementary Figure 3 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells
Supplementary Figure 3 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells Open
PDF file - 20K, Comparative WNT transcriptional activity and growth inhibition with different compound AXIN stabilizers.
View article: Supplementary Figure 6 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells
Supplementary Figure 6 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells Open
PDF file - 112K, Average body weight graph.
View article: Supplementary Figure 5 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells
Supplementary Figure 5 from Identification and Characterization of a Small-Molecule Inhibitor of Wnt Signaling in Glioblastoma Cells Open
PDF file - 98K, In vitro response of T98G cells to WNT signaling inhibition.
View article: Antiviral activities of peptide-based covalent inhibitors of the Enterovirus 71 3C protease
Antiviral activities of peptide-based covalent inhibitors of the Enterovirus 71 3C protease Open
Hand, Foot and Mouth Disease is a highly contagious disease caused by a range of human enteroviruses. Outbreaks occur regularly, especially in the Asia-Pacific region, putting a burden on public healthcare systems. Currently, there is no a…
View article: Escherichia coli Topoisomerase IV E Subunit and an Inhibitor Binding Mode Revealed by NMR Spectroscopy
Escherichia coli Topoisomerase IV E Subunit and an Inhibitor Binding Mode Revealed by NMR Spectroscopy Open
Bacterial topoisomerases are attractive antibacterial drug targets because of their importance in bacterial growth and low homology with other human topoisomerases. Structure-based drug design has been a proven approach of efficiently deve…
View article: Peptidomimetic ethyl propenoate covalent inhibitors of the enterovirus 71 3C protease: a P2–P4 study
Peptidomimetic ethyl propenoate covalent inhibitors of the enterovirus 71 3C protease: a P2–P4 study Open
View article: Peptidomimetic ethyl propenoate covalent inhibitors of the enterovirus 71 3C protease: a P2–P4 study
Peptidomimetic ethyl propenoate covalent inhibitors of the enterovirus 71 3C protease: a P2–P4 study Open
View article: Peptidomimetic ethyl propenoate covalent inhibitors of the enterovirus 71 3C protease: a P2–P4 study
Peptidomimetic ethyl propenoate covalent inhibitors of the enterovirus 71 3C protease: a P2–P4 study Open
Enterovirus 71 (EV71) is a highly infectious pathogen primarily responsible for Hand, Foot, and Mouth Disease, particularly among children. Currently, no approved antiviral drug has been developed against this disease. The EV71 3C protease…