Sophie Vasseur
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View article: The HDAC inhibitor romidepsin renders liver cancer vulnerable to RTK targeting and immunologically active
The HDAC inhibitor romidepsin renders liver cancer vulnerable to RTK targeting and immunologically active Open
View article: The extracellular matrix drives guanylate production and protects pancreatic cancer cells from oxaliplatin-induced DNA damage
The extracellular matrix drives guanylate production and protects pancreatic cancer cells from oxaliplatin-induced DNA damage Open
The excessive production of extracellular matrix (ECM) and the metabolic adaptations in pancreatic ductal adenocarcinoma (PDAC) contribute individually to enhanced chemoresistance, dramatic tumor progression and dismal patient survival. Ho…
View article: Functional and Molecular Characterization of New SPTLC1 Missense Variants in Patients with Hereditary Sensory and Autonomic Neuropathy Type 1 (HSAN1)
Functional and Molecular Characterization of New SPTLC1 Missense Variants in Patients with Hereditary Sensory and Autonomic Neuropathy Type 1 (HSAN1) Open
Hereditary sensory and autonomic neuropathy type 1 is an autosomal dominant neuropathy caused by the SPTLC1 or SPTLC2 variants. These variants modify the preferred substrate of serine palmitoyl transferase, responsible for the first step o…
View article: Supplementary Table 2 from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A
Supplementary Table 2 from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A Open
Primer pairs used for real-time quantitative RT-PCR.
View article: Figure S1 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker
Figure S1 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker Open
Measurement of LIF mRNA level in normal and tumoral human fibroblasts by RT-QPCRs.
View article: Supplementary Table 1 from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A
Supplementary Table 1 from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A Open
Supplemental Table 1. List of used Primary antibodies.
View article: Figure S2 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker
Figure S2 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker Open
Schematic representation of experimental protocols for (A) the co-culture method in order to produce specific conditioned media used in following migration assay or ELISA tittering as for (B) the migration assay method for the measurement …
View article: Supplementary Table 2 from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A
Supplementary Table 2 from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A Open
Primer pairs used for real-time quantitative RT-PCR.
View article: Data from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker
Data from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker Open
Pancreatic ductal adenocarcinoma (PDAC) is characterized by extensive stroma and pathogenic modifications to the peripheral nervous system that elevate metastatic capacity. In this study, we show that the IL6-related stem cell–promoting fa…
View article: Data from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A
Data from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A Open
Pancreatic ductal adenocarcinoma (PDAC) is a deadly and aggressive cancer. Understanding mechanisms that drive preneoplastic pancreatic lesions is necessary to improve early diagnostic and therapeutic strategies. Mutations and inactivation…
View article: Figure S4 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker
Figure S4 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker Open
We analyzed the impact of LIF (A) or conditioned media (B) on sNF96.2 cell survival using caspase-3 positive cell measurement.
View article: Figure S1 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker
Figure S1 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker Open
Measurement of LIF mRNA level in normal and tumoral human fibroblasts by RT-QPCRs.
View article: Figure S4 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker
Figure S4 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker Open
We analyzed the impact of LIF (A) or conditioned media (B) on sNF96.2 cell survival using caspase-3 positive cell measurement.
View article: Data from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A
Data from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A Open
Pancreatic ductal adenocarcinoma (PDAC) is a deadly and aggressive cancer. Understanding mechanisms that drive preneoplastic pancreatic lesions is necessary to improve early diagnostic and therapeutic strategies. Mutations and inactivation…
View article: Figure S2 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker
Figure S2 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker Open
Schematic representation of experimental protocols for (A) the co-culture method in order to produce specific conditioned media used in following migration assay or ELISA tittering as for (B) the migration assay method for the measurement …
View article: Figure S3 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker
Figure S3 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker Open
Amalysis of Akt/Stat3 signaling induction following LIF stimulation. We measured the induction of STAT3 (A) and Akt (B) phosphorylation by western blot in sNF96.2 after incubation with LIF recombinant protein. We confirmed the specificity …
View article: Data from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker
Data from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker Open
Pancreatic ductal adenocarcinoma (PDAC) is characterized by extensive stroma and pathogenic modifications to the peripheral nervous system that elevate metastatic capacity. In this study, we show that the IL6-related stem cell–promoting fa…
View article: Supplementary Data from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A
Supplementary Data from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A Open
Supplemental Materials & Methods and Figures 1-12. Figure S1: Analysis of mouse WT pancreases, exposed to short-term sActRIIB-Fc treatment. Figure S2: Inactivation of Acvr1b, combined with oncogenic Kras, results in the development of enla…
View article: Figure S3 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker
Figure S3 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker Open
Amalysis of Akt/Stat3 signaling induction following LIF stimulation. We measured the induction of STAT3 (A) and Akt (B) phosphorylation by western blot in sNF96.2 after incubation with LIF recombinant protein. We confirmed the specificity …
View article: Supplementary Material & figure legends from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A
Supplementary Material & figure legends from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A Open
Supplementary Material & figure legends
View article: Figure S5 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker
Figure S5 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker Open
Measurement of Schwann cell differentiation through JAK/STAT pathway following LIF induction. The staining of 2 differentiation markers (S100 and Pou3F2) were validated in human PDA samples (A) then used on sNF96.2 cells following conditio…
View article: Supplementary Table 1 from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A
Supplementary Table 1 from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A Open
Supplemental Table 1. List of used Primary antibodies.
View article: Figure S5 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker
Figure S5 from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker Open
Measurement of Schwann cell differentiation through JAK/STAT pathway following LIF induction. The staining of 2 differentiation markers (S100 and Pou3F2) were validated in human PDA samples (A) then used on sNF96.2 cells following conditio…
View article: Supplementary data from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker
Supplementary data from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker Open
Supplementary methods and Figure legends
View article: Supplementary Data from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A
Supplementary Data from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A Open
Supplemental Materials & Methods and Figures 1-12. Figure S1: Analysis of mouse WT pancreases, exposed to short-term sActRIIB-Fc treatment. Figure S2: Inactivation of Acvr1b, combined with oncogenic Kras, results in the development of enla…
View article: Supplementary Material & figure legends from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A
Supplementary Material & figure legends from Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A Open
Supplementary Material & figure legends
View article: Supplementary data from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker
Supplementary data from LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker Open
Supplementary methods and Figure legends
View article: Supplementary Figures 1-6 from Tumor Protein 53–Induced Nuclear Protein 1 Is a Major Mediator of p53 Antioxidant Function
Supplementary Figures 1-6 from Tumor Protein 53–Induced Nuclear Protein 1 Is a Major Mediator of p53 Antioxidant Function Open
Supplementary Figures 1-6 from Tumor Protein 53–Induced Nuclear Protein 1 Is a Major Mediator of p53 Antioxidant Function
View article: Supplementary Figure Legends 1-6 from Tumor Protein 53–Induced Nuclear Protein 1 Is a Major Mediator of p53 Antioxidant Function
Supplementary Figure Legends 1-6 from Tumor Protein 53–Induced Nuclear Protein 1 Is a Major Mediator of p53 Antioxidant Function Open
Supplementary Figure Legends 1-6 from Tumor Protein 53–Induced Nuclear Protein 1 Is a Major Mediator of p53 Antioxidant Function
View article: Supplementary Methods from Tumor Protein 53–Induced Nuclear Protein 1 Is a Major Mediator of p53 Antioxidant Function
Supplementary Methods from Tumor Protein 53–Induced Nuclear Protein 1 Is a Major Mediator of p53 Antioxidant Function Open
Supplementary Methods from Tumor Protein 53–Induced Nuclear Protein 1 Is a Major Mediator of p53 Antioxidant Function