Sumiko Kohashi
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View article: Aberrant CD4 expression in plasmablastic transformation of multiple myeloma
Aberrant CD4 expression in plasmablastic transformation of multiple myeloma Open
A 68-year-old female patient with a 5-year history of λ-type IgD plasmacytoma was admitted to our hospital for the evaluation of fatigue in September 2023. The diagnosis of plasmacytoma was confirmed through a biopsy of the bone mass lesio…
View article: Daratumumab improved severe neutrophilia and paraneoplastic bone marrow fibrosis in granulocyte‐colony stimulating factor‐producing multiple myeloma
Daratumumab improved severe neutrophilia and paraneoplastic bone marrow fibrosis in granulocyte‐colony stimulating factor‐producing multiple myeloma Open
In cases of unexplained neutrophilia, granulocyte-colony stimulating factor (G-CSF)-producing tumours should be considered. Although such tumours are mainly reported in solid cancers, a few cases of G-CSF-producing multiple myeloma (MM) ha…
View article: Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C
Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C Open
When the etiology of pancreatitis cannot be determined despite sufficient investigation, recurrence and progression to chronic pancreatitis often involve genetic mutations. Herein, we describe a case of recurrent pancreatitis with the IVS3…
View article: Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C
Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C Open
When the etiology of pancreatitis cannot be determined despite sufficient investigation, recurrence and progression to chronic pancreatitis often involve genetic mutations. Herein, we describe a case of recurrent pancreatitis with the IVS3…
View article: Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C
Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C Open
When the etiology of pancreatitis cannot be determined despite sufficient investigation, recurrence and progression to chronic pancreatitis often involve genetic mutations. Herein, we describe a case of recurrent pancreatitis with the IVS3…
View article: Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C
Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C Open
When the etiology of pancreatitis cannot be determined despite sufficient investigation, recurrence and progression to chronic pancreatitis often involve genetic mutations. Herein, we describe a case of recurrent pancreatitis with the IVS3…
View article: Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C
Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C Open
When the etiology of pancreatitis cannot be determined despite sufficient investigation, recurrence and progression to chronic pancreatitis often involve genetic mutations. Herein, we describe a case of recurrent pancreatitis with the IVS3…
View article: Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C
Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C Open
When the etiology of pancreatitis cannot be determined despite sufficient investigation, recurrence and progression to chronic pancreatitis often involve genetic mutations. Herein, we describe a case of recurrent pancreatitis with the IVS3…
View article: Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C
Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C Open
When the etiology of pancreatitis cannot be determined despite sufficient investigation, recurrence and progression to chronic pancreatitis often involve genetic mutations. Herein, we describe a case of recurrent pancreatitis with the IVS3…
View article: Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C
Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C Open
When the etiology of pancreatitis cannot be determined despite sufficient investigation, recurrence and progression to chronic pancreatitis often involve genetic mutations. Herein, we describe a case of recurrent pancreatitis with the IVS3…
View article: Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C
Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C Open
When the etiology of pancreatitis cannot be determined despite sufficient investigation, recurrence and progression to chronic pancreatitis often involve genetic mutations. Herein, we describe a case of recurrent pancreatitis with the IVS3…
View article: Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C
Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C Open
When the etiology of pancreatitis cannot be determined despite sufficient investigation, recurrence and progression to chronic pancreatitis often involve genetic mutations. Herein, we describe a case of recurrent pancreatitis with the IVS3…
View article: Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C
Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C Open
When the etiology of pancreatitis cannot be determined despite sufficient investigation, recurrence and progression to chronic pancreatitis often involve genetic mutations. Herein, we describe a case of recurrent pancreatitis with the IVS3…
View article: Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C
Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C Open
When the etiology of pancreatitis cannot be determined despite sufficient investigation, recurrence and progression to chronic pancreatitis often involve genetic mutations. Herein, we describe a case of recurrent pancreatitis with the IVS3…
View article: Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C
Supplementary Material for: Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to <i>SPINK1</i> Mutation IVS3+2T>C Open
When the etiology of pancreatitis cannot be determined despite sufficient investigation, recurrence and progression to chronic pancreatitis often involve genetic mutations. Herein, we describe a case of recurrent pancreatitis with the IVS3…
View article: Successful treatment of breakthrough disseminated Trichosporon asahii fungemia in a patient with acute myeloid leukemia receiving itraconazole prophylaxis
Successful treatment of breakthrough disseminated Trichosporon asahii fungemia in a patient with acute myeloid leukemia receiving itraconazole prophylaxis Open
We encountered a case of a 73-year-old man with acute myeloid leukemia who developed Trichosporon asahii systemic infection while on itraconazole prophylaxis during severe neutropenia. Cryptococcal antigen was useful for diagnosis. Althoug…
View article: REAL‐LIFE USE OF BENDAMUSTINE FOR B‐CELL NON‐HODGKIN LYMPHOMA IN A COMMUNITY HOSPITAL IN JAPAN—RETREATMENT WITH BENDAMUSTINE IS SAFE AND FEASIBLE
REAL‐LIFE USE OF BENDAMUSTINE FOR B‐CELL NON‐HODGKIN LYMPHOMA IN A COMMUNITY HOSPITAL IN JAPAN—RETREATMENT WITH BENDAMUSTINE IS SAFE AND FEASIBLE Open
Introduction: Bendamustine is an alkylating agent with antimetabolite properties that has little cross resistance to other alkylating agents and purine analogues. It has shown to be effective in treatment of relapsed/refractory (R/R) chron…
View article: Long-Term Follow-up of Allogeneic Hematopoietic Stem Cell Transplantation for Follicular Lymphoma and Clinical Evidence of Graft-Versus-Lymphoma Effect in Post-Transplant Relapsed Patients
Long-Term Follow-up of Allogeneic Hematopoietic Stem Cell Transplantation for Follicular Lymphoma and Clinical Evidence of Graft-Versus-Lymphoma Effect in Post-Transplant Relapsed Patients Open
Allogeneic hematopoietic stem cell transplantation (HSCT) has been established as a curative therapy for refractory or relapsed follicular lymphoma. This promising effect is considered due to graft-versus-lymphoma (GVL) effect, and several…