Thiago Belchior
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View article: Chloroquine Attenuates Diet-Induced Obesity and Glucose Intolerance Through a Mechanism that Might Involve FGF-21, But not UCP-1-Mediated Thermogenesis and Inhibition of Adipocyte Autophagy
Chloroquine Attenuates Diet-Induced Obesity and Glucose Intolerance Through a Mechanism that Might Involve FGF-21, But not UCP-1-Mediated Thermogenesis and Inhibition of Adipocyte Autophagy Open
View article: PPARγ-induced upregulation of subcutaneous fat adiponectin secretion, glyceroneogenesis and BCAA oxidation requires mTORC1 activity
PPARγ-induced upregulation of subcutaneous fat adiponectin secretion, glyceroneogenesis and BCAA oxidation requires mTORC1 activity Open
View article: Obesity-Linked PPARγ S273 Phosphorylation Promotes Insulin Resistance through Growth Differentiation Factor 3
Obesity-Linked PPARγ S273 Phosphorylation Promotes Insulin Resistance through Growth Differentiation Factor 3 Open
View article: Palmitoleic acid reduces high fat diet-induced liver inflammation by promoting PPAR-γ-independent M2a polarization of myeloid cells
Palmitoleic acid reduces high fat diet-induced liver inflammation by promoting PPAR-γ-independent M2a polarization of myeloid cells Open
View article: Obesity-linked PPARγ S273 phosphorylation promotes insulin resistance through Growth Differentiation Factor 3
Obesity-linked PPARγ S273 phosphorylation promotes insulin resistance through Growth Differentiation Factor 3 Open
Overnutrition and obesity promote adipose tissue dysfunction, often leading to systemic insulin resistance. The thiazolidinediones (TZDs) are a potent class of insulin-sensitizing drugs and ligands of PPARγ that improve insulin sensitivity…
View article: Regulation of Metabolic Disease-Associated Inflammation by Nutrient Sensors
Regulation of Metabolic Disease-Associated Inflammation by Nutrient Sensors Open
Visceral obesity is frequently associated with the development of type 2 diabetes (T2D), a highly prevalent chronic disease that features insulin resistance and pancreatic β -cell dysfunction as important hallmarks. Recent evidence indicat…
View article: Cagaita fruit ( Eugenia dysenterica DC.) and obesity: Role of polyphenols on already established obesity
Cagaita fruit ( Eugenia dysenterica DC.) and obesity: Role of polyphenols on already established obesity Open
View article: Author Index
Author Index Open
View article: Adipocyte mTORC1 deficiency promotes adipose tissue inflammation and NLRP3 inflammasome activation via oxidative stress and de novo ceramide synthesis
Adipocyte mTORC1 deficiency promotes adipose tissue inflammation and NLRP3 inflammasome activation via oxidative stress and de novo ceramide synthesis Open
View article: Dectin-1 Activation Exacerbates Obesity and Insulin Resistance in the Absence of MyD88
Dectin-1 Activation Exacerbates Obesity and Insulin Resistance in the Absence of MyD88 Open
View article: mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function
mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function Open
View article: Constitutive adipocyte mTORC1 activation enhances mitochondrial activity and reduces visceral adiposity in mice
Constitutive adipocyte mTORC1 activation enhances mitochondrial activity and reduces visceral adiposity in mice Open
View article: Phenolic compounds from cagaita (Eugenia dysenterica DC.) fruit prevent body weight and fat mass gain induced by a high-fat, high-sucrose diet
Phenolic compounds from cagaita (Eugenia dysenterica DC.) fruit prevent body weight and fat mass gain induced by a high-fat, high-sucrose diet Open
View article: Omega‐3 fatty acids protect from diet‐induced obesity, glucose intolerance, and adipose tissue inflammation through PPARγ‐dependent and PPARγ‐independent actions
Omega‐3 fatty acids protect from diet‐induced obesity, glucose intolerance, and adipose tissue inflammation through PPARγ‐dependent and PPARγ‐independent actions Open
Scope We tested herein the hypothesis that peroxisome proliferator activated receptor γ (PPARγ) is a major mediator of omega‐3 ( n ‐3) protective actions against high‐fat diet (HFD) induced obesity, glucose intolerance, and adipose tissue …