Timothy Marsh
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View article: Supplementary Figure 8 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells
Supplementary Figure 8 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells Open
PDF file - 89K, Aspirin treatment does not affect growth kinetics of the LBC instigator or circulating platelet counts
View article: Supplementary Figure 5 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells
Supplementary Figure 5 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells Open
PDF file - 490K, Images used for quantification of responding tumor vessel density under indicated systemic environments; analysis reveals enhanced vascularization in the instigating LBC environment
View article: Supplementary Figure 8 from Stromal EGF and IGF-I Together Modulate Plasticity of Disseminated Triple-Negative Breast Tumors
Supplementary Figure 8 from Stromal EGF and IGF-I Together Modulate Plasticity of Disseminated Triple-Negative Breast Tumors Open
PDF file - 159K, Identification of Instigating Human Breast Tumor Surgical Specimens.
View article: Supplementary Figure 1 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells
Supplementary Figure 1 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells Open
PDF file - 287K, Characterization of responding tumors in LBC and TNBC systemic environments suggests different mechanisms of instigation
View article: Data from p38MAPK Plays a Crucial Role in Stromal-Mediated Tumorigenesis
Data from p38MAPK Plays a Crucial Role in Stromal-Mediated Tumorigenesis Open
Neoplastic cells rely on the tumor microenvironment (TME) for survival and progression factors. Indeed, senescent and cancer-associated fibroblasts (CAF) express factors that promote tumorigenesis that are collectively referred to as the s…
View article: Supplementary Figure Legends from p38MAPK Plays a Crucial Role in Stromal-Mediated Tumorigenesis
Supplementary Figure Legends from p38MAPK Plays a Crucial Role in Stromal-Mediated Tumorigenesis Open
PDF file 62K, legends for supplemental figures
View article: Supplementary Figure 3 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells
Supplementary Figure 3 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells Open
PDF file - 166K, Efficiency of BMC transplantation and tissue recruitment of BMCs; effects of LBC environment on responding tumor cell CD24 enrichment and circulating platelet counts
View article: Supplementary Methods, Figure Legend, Tables 1 - 2 from Stromal EGF and IGF-I Together Modulate Plasticity of Disseminated Triple-Negative Breast Tumors
Supplementary Methods, Figure Legend, Tables 1 - 2 from Stromal EGF and IGF-I Together Modulate Plasticity of Disseminated Triple-Negative Breast Tumors Open
PDF file - 125K, Supplemental Table 1: PCR Oligonucleotide Primers. Supplemental Table 2: Antibodies.
View article: Supplementary Figure 1 from Stromal EGF and IGF-I Together Modulate Plasticity of Disseminated Triple-Negative Breast Tumors
Supplementary Figure 1 from Stromal EGF and IGF-I Together Modulate Plasticity of Disseminated Triple-Negative Breast Tumors Open
PDF file - 181K, Instigating Tumors Facilitate Responder Growth in the Absence of "Self-seeding".
View article: Supplementary Table 1 from p38MAPK Plays a Crucial Role in Stromal-Mediated Tumorigenesis
Supplementary Table 1 from p38MAPK Plays a Crucial Role in Stromal-Mediated Tumorigenesis Open
PDF file 401K, List of p38MAPK-dependent SASP factors and their overlap with publically available expression analyses from breast cancer-associated microenvironments
View article: Supplementary Figure 7 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells
Supplementary Figure 7 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells Open
PDF file - 117K, Tumor specimen from a patient with with clear cell renal cell carcinoma responders to the LBC environment in the systemic instigation xenograft model
View article: Supplementary Figure 7 from Stromal EGF and IGF-I Together Modulate Plasticity of Disseminated Triple-Negative Breast Tumors
Supplementary Figure 7 from Stromal EGF and IGF-I Together Modulate Plasticity of Disseminated Triple-Negative Breast Tumors Open
PDF file - 279K, EGFR/IGFR Inhibitors do not Affect the Phenotype of Responding Tumor Cells in Animals Bearing Control Matrigel.
View article: Supplementary Figure 7 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells
Supplementary Figure 7 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells Open
PDF file - 117K, Tumor specimen from a patient with with clear cell renal cell carcinoma responders to the LBC environment in the systemic instigation xenograft model
View article: Data from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells
Data from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells Open
Breast cancer recurrence rates vary following treatment, suggesting that tumor cells disseminate early from primary sites but remain indolent indefinitely before progressing to symptomatic disease. The reasons why some indolent disseminate…
View article: Supplementary Figure 5 from Stromal EGF and IGF-I Together Modulate Plasticity of Disseminated Triple-Negative Breast Tumors
Supplementary Figure 5 from Stromal EGF and IGF-I Together Modulate Plasticity of Disseminated Triple-Negative Breast Tumors Open
PDF file - 51K, EGF and IGF-1 Together Modulate Tumorogenicity of Responder Cells in vitro.
View article: Supplementary Figure 3 from p38MAPK Plays a Crucial Role in Stromal-Mediated Tumorigenesis
Supplementary Figure 3 from p38MAPK Plays a Crucial Role in Stromal-Mediated Tumorigenesis Open
PDF file 229K, qRT-PCR validation of RNA-seq analysis and TNFalpha ELISA on p38i treated mice
View article: Supplementary Table 1 from p38MAPK Plays a Crucial Role in Stromal-Mediated Tumorigenesis
Supplementary Table 1 from p38MAPK Plays a Crucial Role in Stromal-Mediated Tumorigenesis Open
PDF file 401K, List of p38MAPK-dependent SASP factors and their overlap with publically available expression analyses from breast cancer-associated microenvironments
View article: Data from p38MAPK Plays a Crucial Role in Stromal-Mediated Tumorigenesis
Data from p38MAPK Plays a Crucial Role in Stromal-Mediated Tumorigenesis Open
Neoplastic cells rely on the tumor microenvironment (TME) for survival and progression factors. Indeed, senescent and cancer-associated fibroblasts (CAF) express factors that promote tumorigenesis that are collectively referred to as the s…
View article: Supplementary Figure 8 from Stromal EGF and IGF-I Together Modulate Plasticity of Disseminated Triple-Negative Breast Tumors
Supplementary Figure 8 from Stromal EGF and IGF-I Together Modulate Plasticity of Disseminated Triple-Negative Breast Tumors Open
PDF file - 159K, Identification of Instigating Human Breast Tumor Surgical Specimens.
View article: Supplementary Figure 4 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells
Supplementary Figure 4 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells Open
PDF file - 214K, The LBC environment promotes pro-angiogenic cytokine enrichment in circulating platelets but does not significantly affect phospho-STAT3 levels in BMCs
View article: Supplementary Methods, Figure Legends 1-8 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells
Supplementary Methods, Figure Legends 1-8 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells Open
PDF file - 90K
View article: Supplementary Figure 2 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells
Supplementary Figure 2 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells Open
PDF file - 264K, Contributions of different vessel cell types to responding tumor vasculature under different instigating systemic environments
View article: Supplementary Figure 3 from p38MAPK Plays a Crucial Role in Stromal-Mediated Tumorigenesis
Supplementary Figure 3 from p38MAPK Plays a Crucial Role in Stromal-Mediated Tumorigenesis Open
PDF file 229K, qRT-PCR validation of RNA-seq analysis and TNFalpha ELISA on p38i treated mice
View article: Supplementary Figure 2 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells
Supplementary Figure 2 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells Open
PDF file - 264K, Contributions of different vessel cell types to responding tumor vasculature under different instigating systemic environments
View article: Supplementary Figure 7 from Stromal EGF and IGF-I Together Modulate Plasticity of Disseminated Triple-Negative Breast Tumors
Supplementary Figure 7 from Stromal EGF and IGF-I Together Modulate Plasticity of Disseminated Triple-Negative Breast Tumors Open
PDF file - 279K, EGFR/IGFR Inhibitors do not Affect the Phenotype of Responding Tumor Cells in Animals Bearing Control Matrigel.
View article: Supplementary Figure 1 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells
Supplementary Figure 1 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells Open
PDF file - 287K, Characterization of responding tumors in LBC and TNBC systemic environments suggests different mechanisms of instigation
View article: Supplementary Figure 5 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells
Supplementary Figure 5 from Identification of Luminal Breast Cancers That Establish a Tumor-Supportive Macroenvironment Defined by Proangiogenic Platelets and Bone Marrow–Derived Cells Open
PDF file - 490K, Images used for quantification of responding tumor vessel density under indicated systemic environments; analysis reveals enhanced vascularization in the instigating LBC environment
View article: Supplementary Figure 2 from p38MAPK Plays a Crucial Role in Stromal-Mediated Tumorigenesis
Supplementary Figure 2 from p38MAPK Plays a Crucial Role in Stromal-Mediated Tumorigenesis Open
PDF file 545K, SASP upregulation following bleomycin, NaB or replication-induced senescence. SASP mRNA stabilization in NaB-treated BJs and in bleomycin-treated IMR90s
View article: Supplementary Figure 5 from Stromal EGF and IGF-I Together Modulate Plasticity of Disseminated Triple-Negative Breast Tumors
Supplementary Figure 5 from Stromal EGF and IGF-I Together Modulate Plasticity of Disseminated Triple-Negative Breast Tumors Open
PDF file - 51K, EGF and IGF-1 Together Modulate Tumorogenicity of Responder Cells in vitro.