Trygve Andreassen
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View article: Unaltered 3’-sialyllactose and 6’-sialyllactose concentrations in human milk acutely after endurance exercise: a randomized crossover trial
Unaltered 3’-sialyllactose and 6’-sialyllactose concentrations in human milk acutely after endurance exercise: a randomized crossover trial Open
Introduction Human milk contains over 200 different types of human milk oligosaccharides (HMOs) with concentrations varying based on genetics, lifestyle, and time postpartum. Prior research indicates that exercise training during pregnancy…
View article: Stability in fecal metabolites amid a diverse gut microbiome composition: a one-month longitudinal study of variability in healthy individuals
Stability in fecal metabolites amid a diverse gut microbiome composition: a one-month longitudinal study of variability in healthy individuals Open
An extensive network of microbial-host interactions exists in the gut, making the gut microbiome a complex ecosystem to untangle. The microbial composition and the fecal metabolites are important readouts to investigate intricate microbiot…
View article: Serum Lipoprotein Profiling by NMR Spectroscopy Reveals Alterations in HDL-1 and HDL-2 Apo-A2 Subfractions in Alzheimer’s Disease
Serum Lipoprotein Profiling by NMR Spectroscopy Reveals Alterations in HDL-1 and HDL-2 Apo-A2 Subfractions in Alzheimer’s Disease Open
Identifying biomarkers for Alzheimer’s disease (AD) is crucial, due to its complex pathology, which involves dysfunction in lipid transport, contributing to neuroinflammation, synaptic loss, and impaired amyloid-β clearance. Nuclear magnet…
View article: Identifying possible biomarkers of lower urinary tract symptoms using metabolomics and partial least square regression
Identifying possible biomarkers of lower urinary tract symptoms using metabolomics and partial least square regression Open
Introduction The objective of this study was to explore potential novel biomarkers for moderate to severe lower urinary tract symptoms (LUTS) using a metabolomics-based approach, and statistical methods with significant different features …
View article: Serum NMR-Based Metabolomics Profiling Identifies Lipoprotein Subfraction Variables and Amino Acid Reshuffling in Myeloma Development and Progression
Serum NMR-Based Metabolomics Profiling Identifies Lipoprotein Subfraction Variables and Amino Acid Reshuffling in Myeloma Development and Progression Open
Multiple myeloma (MM) is an incurable hematological cancer. It is preceded by monoclonal gammopathy of uncertain significance (MGUS)—an asymptomatic phase. It has been demonstrated that early detection increases the 5-year survival rate. H…
View article: Serum NMR-Based Metabolomics Profiling Identifies Lipoprotein Subfraction Variables and Amino Acid Reshuffling in Myeloma Development and Progression
Serum NMR-Based Metabolomics Profiling Identifies Lipoprotein Subfraction Variables and Amino Acid Reshuffling in Myeloma Development and Progression Open
Multiple myeloma (MM) is an incurable hematological cancer. It is preceded by monoclonal gammopathy of uncertain significance (MGUS), an asymptomatic phase. It has been demonstrated that early detection increases the 5-year survival rate. …
View article: Serum metabolic signatures for Alzheimer’s Disease reveal alterations in amino acid composition and energy metabolism – A validation study
Serum metabolic signatures for Alzheimer’s Disease reveal alterations in amino acid composition and energy metabolism – A validation study Open
Background: Alzheimer’s Disease (AD) is complex and novel approaches are urgently needed to characterise disease pathology and to aid in diagnosis. Metabolites are the end-products of upstream molecular alterations, whereby small changes a…
View article: Data from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Data from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
Estrogen receptor α (ERα) is a key regulator of breast growth and breast cancer development. Here, we report how ERα impacts these processes by reprogramming metabolism in malignant breast cells. We employed an integrated approach, combini…
View article: Supplementary figure 1 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary figure 1 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
Characterization of ERα cistromes for MCF7 and T47D cells
View article: Supplementary table 5 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary table 5 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
Microarray and qPCR data showing no induction of PEMT mRNA levels by estrogen in MCF7 and T47D cells
View article: Data from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Data from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
Estrogen receptor α (ERα) is a key regulator of breast growth and breast cancer development. Here, we report how ERα impacts these processes by reprogramming metabolism in malignant breast cells. We employed an integrated approach, combini…
View article: Supplementary table 6 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary table 6 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
Association between CHPT1 expression in breast cancer samples and clinicopathological data.
View article: Supplementary table 1 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary table 1 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
The core set of direct ERalpha regulated genes in breast cancer cells
View article: Supplementary table 2 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary table 2 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
The metabolites levels identified by NMR, which are normalized by protein amount
View article: Supplementary table 1 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary table 1 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
The core set of direct ERalpha regulated genes in breast cancer cells
View article: Supplementary table 3 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary table 3 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
Changed levels of intracellular metabolites in MCF7 and T47 D cells in response to estrogen
View article: Supplementary Materials and Methods and Supplementary Figure and Table Legends from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary Materials and Methods and Supplementary Figure and Table Legends from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
Supplementary Materials and Methods and Supplementary Figure and Table Legends
View article: Supplementary figure 2 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary figure 2 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
Metabolic profiles of breast cancer cells in response to estrogen treatment
View article: Supplementary table 2 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary table 2 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
The metabolites levels identified by NMR, which are normalized by protein amount
View article: Supplementary table 5 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary table 5 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
Microarray and qPCR data showing no induction of PEMT mRNA levels by estrogen in MCF7 and T47D cells
View article: Supplementary table 4 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary table 4 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
Changes of Cho-containing metabolites upon E2 stimulation
View article: Supplementary table 4 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary table 4 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
Changes of Cho-containing metabolites upon E2 stimulation
View article: Supplementary table 6 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary table 6 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
Association between CHPT1 expression in breast cancer samples and clinicopathological data.
View article: Supplementary figure 1 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary figure 1 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
Characterization of ERα cistromes for MCF7 and T47D cells
View article: Supplementary table 3 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary table 3 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
Changed levels of intracellular metabolites in MCF7 and T47 D cells in response to estrogen
View article: Supplementary figure 2 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary figure 2 from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
Metabolic profiles of breast cancer cells in response to estrogen treatment
View article: Supplementary Materials and Methods and Supplementary Figure and Table Legends from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism
Supplementary Materials and Methods and Supplementary Figure and Table Legends from Estrogen Receptor α Promotes Breast Cancer by Reprogramming Choline Metabolism Open
Supplementary Materials and Methods and Supplementary Figure and Table Legends