Urmi Bagchi
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View article: 387P Histone deacetylase 6 inhibition abrogates the STAT3-mediated protumoral polarization of M2 TAMs and suppresses the VEGFA/VEGFR2-induced macrophage-breast cancer crosstalk in the tumor microenvironment
387P Histone deacetylase 6 inhibition abrogates the STAT3-mediated protumoral polarization of M2 TAMs and suppresses the VEGFA/VEGFR2-induced macrophage-breast cancer crosstalk in the tumor microenvironment Open
View article: Inhibition of STAT3 by 2-Methoxyestradiol suppresses M2 polarization and protumoral functions of macrophages in breast cancer
Inhibition of STAT3 by 2-Methoxyestradiol suppresses M2 polarization and protumoral functions of macrophages in breast cancer Open
View article: 177P The immune-microenvironment confers chemoresistance in breast cancer through activation of VEGFR2/STAT3/BIRC5 signaling
177P The immune-microenvironment confers chemoresistance in breast cancer through activation of VEGFR2/STAT3/BIRC5 signaling Open
Chemotherapy with Taxanes is often the preferred clinical treatment for breast cancer patients. Unfortunately, these agents become increasingly ineffective during treatment. Pro-tumoral M2 tumor-associated macrophages (TAMs) are the most a…
View article: Inhibition of STAT3 by 2-Methoxyestradiol suppresses M2 polarization and protumoral functions of macrophages in breast cancer
Inhibition of STAT3 by 2-Methoxyestradiol suppresses M2 polarization and protumoral functions of macrophages in breast cancer Open
Background Breast cancer metastasis remains the leading cause of cancer-related deaths in women worldwide. Infiltration of tumor-associated macrophages (TAMs) in the tumor stroma is known to be correlated with reduced overall survival, and…