Weisan Chen
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View article: Early boosting of p38 MAPK signaling pathway by lycorine hydrochloride potently inhibits PRRSV proliferation in primary and established cells
Early boosting of p38 MAPK signaling pathway by lycorine hydrochloride potently inhibits PRRSV proliferation in primary and established cells Open
Porcine reproductive and respiratory syndrome virus (PRRSV) has caused tremendous economic losses in the swine industry since emerging in the late 1980s. Although vaccination has been widely used to control PRRS epidemics in Chinese pig fa…
View article: Betulonic Acid Inhibits Type-2 Porcine Reproductive and Respiratory Syndrome Virus Replication by Downregulating Cellular ATP Production
Betulonic Acid Inhibits Type-2 Porcine Reproductive and Respiratory Syndrome Virus Replication by Downregulating Cellular ATP Production Open
Porcine reproductive and respiratory syndrome (PRRS), caused by PRRS virus (PRRSV) infection, has been a serious threat to the pork industry worldwide and continues to bring significant economic loss. Current vaccination strategies offer l…
View article: Fangchinoline Inhibits African Swine Fever Virus Replication by Suppressing the AKT/mTOR/NF-κB Signaling Pathway in Porcine Alveolar Macrophages
Fangchinoline Inhibits African Swine Fever Virus Replication by Suppressing the AKT/mTOR/NF-κB Signaling Pathway in Porcine Alveolar Macrophages Open
African swine fever (ASF), caused by the African swine fever virus (ASFV), is one of the most important infectious diseases that cause high morbidity and mortality in pigs and substantial economic losses to the pork industry of affected co…
View article: CD39hi identifies an exhausted tumor-reactive CD8+ T cell population associated with tumor progression in human gastric cancer
CD39hi identifies an exhausted tumor-reactive CD8+ T cell population associated with tumor progression in human gastric cancer Open
The ectonucleotidase CD39 has been regarded as a promising immune checkpoint in solid tumors. However, the expression of CD39 by tumor-infiltrating CD8+ T cells as well as their potential roles and clinical implications in human gastric ca…
View article: <i>Helicobacter pylori</i>-Induced Angiopoietin-Like 4 Promotes Gastric Bacterial Colonization and Gastritis
<i>Helicobacter pylori</i>-Induced Angiopoietin-Like 4 Promotes Gastric Bacterial Colonization and Gastritis Open
Helicobacter pylori infection is characterized as progressive processes of bacterial persistence and chronic gastritis with features of infiltration of mononuclear cells more than granulocytes in gastric mucosa. Angiopoietin-like 4 (ANGPTL…
View article: CD39 expression defines exhausted CD4<sup>+</sup> T cells associated with poor survival and immune evasion in human gastric cancer
CD39 expression defines exhausted CD4<sup>+</sup> T cells associated with poor survival and immune evasion in human gastric cancer Open
Objectives CD4 + T cell helper and regulatory function in human cancers has been well characterised. However, the definition of tumor‐infiltrating CD4 + T cell exhaustion and how it contributes to the immune response and disease progressio…
View article: Supplementary Table S5 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Supplementary Table S5 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Table S5
View article: Supplementary Table S3 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Supplementary Table S3 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Table S3
View article: Supplementary Table S6 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Supplementary Table S6 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Table S6
View article: Data from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Data from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Antigen recognition by CD8+ T cells is governed by the pool of peptide antigens presented on the cell surface in the context of HLA class I complexes. Studies have shown not only a high degree of plasticity in the immunopeptidome, but also…
View article: Supplementary Table S3 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Supplementary Table S3 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Table S3
View article: Supplementary Figures from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Supplementary Figures from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Supplementary Figures S1-S10
View article: Supplementary Table S5 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Supplementary Table S5 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Table S5
View article: Supplementary Table S6 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Supplementary Table S6 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Table S6
View article: Supplementary Table S4 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Supplementary Table S4 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Table S4
View article: Data from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Data from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Antigen recognition by CD8+ T cells is governed by the pool of peptide antigens presented on the cell surface in the context of HLA class I complexes. Studies have shown not only a high degree of plasticity in the immunopeptidome, but also…
View article: Supplementary Table S4 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Supplementary Table S4 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Table S4
View article: Supplementary Table S2 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Supplementary Table S2 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Table S2
View article: Supplementary Figures from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Supplementary Figures from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Supplementary Figures S1-S10
View article: Supplementary Figure and Table Legends from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Supplementary Figure and Table Legends from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Supplementary figure legends and tables descriptors
View article: Supplementary Table S2 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Supplementary Table S2 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Table S2
View article: Supplementary Table S1 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Supplementary Table S1 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Table S1
View article: Supplementary Figure and Table Legends from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Supplementary Figure and Table Legends from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Supplementary figure legends and tables descriptors
View article: Supplementary Table S1 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma
Supplementary Table S1 from Spliced Peptides and Cytokine-Driven Changes in the Immunopeptidome of Melanoma Open
Table S1
View article: Supplementary Figure Legend from Loss of Host Type-I IFN Signaling Accelerates Metastasis and Impairs NK-cell Antitumor Function in Multiple Models of Breast Cancer
Supplementary Figure Legend from Loss of Host Type-I IFN Signaling Accelerates Metastasis and Impairs NK-cell Antitumor Function in Multiple Models of Breast Cancer Open
Supplementary Figure Legend from Loss of Host Type-I IFN Signaling Accelerates Metastasis and Impairs NK-cell Antitumor Function in Multiple Models of Breast Cancer
View article: Data from L-Plastin Promotes Gastric Cancer Growth and Metastasis in a <i>Helicobacter pylori cagA</i>-ERK-SP1–Dependent Manner
Data from L-Plastin Promotes Gastric Cancer Growth and Metastasis in a <i>Helicobacter pylori cagA</i>-ERK-SP1–Dependent Manner Open
Actin cytoskeleton dynamic rearrangement is required for tumor cell metastasis and is a key characteristic of Helicobacter pylori (H. pylori)-infected host cells. Actin cytoskeleton modulation is coordinated by multiple actin-binding prote…
View article: Supplementary Figures 1 through 7 and Supplementary Tables 1 through 3 from Tumor-Associated Monocytes/Macrophages Impair NK-Cell Function via TGFβ1 in Human Gastric Cancer
Supplementary Figures 1 through 7 and Supplementary Tables 1 through 3 from Tumor-Associated Monocytes/Macrophages Impair NK-Cell Function via TGFβ1 in Human Gastric Cancer Open
Figure S1. NK cell and T cell numbers in the peripheral blood, non-tumor and tumor tissues of GC patients. Figure S2. Expression of activating and inhibitory receptors on NK cells in the peripheral blood, non-tumor and tumor tissues of GC …