Wenli Cui
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View article: Loss of RhoA in microglia disables glycolytic adaptation and impairs spinal cord injury recovery through Arhgap25/HIF-1α pathway
Loss of RhoA in microglia disables glycolytic adaptation and impairs spinal cord injury recovery through Arhgap25/HIF-1α pathway Open
RhoA, a small GTPase, plays a pivotal role in various diseases, including spinal cord injury (SCI). Although RhoA inhibition has been traditionally viewed as beneficial for SCI repair, recent clinical trials of RhoA inhibitors in SCI have …
View article: PIK3CA regulates lactate transport through MCT1,MCT4 in diffuse large B-cell lymphoma
PIK3CA regulates lactate transport through MCT1,MCT4 in diffuse large B-cell lymphoma Open
Background:As a metabolite of glycolysis, lactate can not only provide energy for tumor cells, but also participate in the occurrence and development of tumors, immune escape, metastasis and recurrence through a variety of mechanisms. Howe…
View article: Comparison of clinicopathological features between cerebral cystic and alveolar echinococcosis: analysis of 27 cerebral echinococcosis cases in Xinjiang, China
Comparison of clinicopathological features between cerebral cystic and alveolar echinococcosis: analysis of 27 cerebral echinococcosis cases in Xinjiang, China Open
View article: Mutation landscape in Chinese nodal diffuse large B-cell lymphoma by targeted next generation sequencing and their relationship with clinicopathological characteristics
Mutation landscape in Chinese nodal diffuse large B-cell lymphoma by targeted next generation sequencing and their relationship with clinicopathological characteristics Open
View article: Regulating the particle sizes of NaA molecular sieves toward enhanced heavy metal ion adsorption
Regulating the particle sizes of NaA molecular sieves toward enhanced heavy metal ion adsorption Open
We have developed a novel synthesis method for NaA-type nanomolecular sieves. The study proved that this NaA molecular sieve has considerable adsorption capacity and can be reused, advancing molecular sieves for future commercialization.
View article: Supplementary Figure 1 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Supplementary Figure 1 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
BATF2 regulates targets of c-Jun/AP-1.
View article: Supplementary Figure 2 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Supplementary Figure 2 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
Enhanced expression of MET correlates with progression in human CRC.
View article: Supplementary Figure 6 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Supplementary Figure 6 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
IC50 determination of PF-04217903 and IFN-β in LoVo and SW620 cells.
View article: Data from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Data from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
Purpose: BATF2, a novel IFN-stimulated gene, inhibits tumor cell proliferation, invasion, and migration. The objectives of this study were to determine how BATF2 expression is associated with colorectal cancer progression and…
View article: Supplementary Figure 5 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Supplementary Figure 5 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
BATF2 is an IFN-stimulated gene.
View article: Supplementary Figure 7 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Supplementary Figure 7 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
IFN-β and MET inhibitors enhance apoptosis but inhibit the proliferation of CRC cells.
View article: Supplementary Figure 3 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Supplementary Figure 3 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
siMET transfection arrests proliferation and migration of CRC cells.
View article: Supplementary Figure Legends, Tables 1 - 4 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Supplementary Figure Legends, Tables 1 - 4 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
Supplementary Table 1. The correlation of BATF2 expression with clinicopathological status in 148 patients with colorectal cancer. Supplementary Table 2. Multivariate Cox proportional hazard analysis of prognostic variables in CRC patients…
View article: Supplementary Figure Legends, Tables 1 - 4 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Supplementary Figure Legends, Tables 1 - 4 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
Supplementary Table 1. The correlation of BATF2 expression with clinicopathological status in 148 patients with colorectal cancer. Supplementary Table 2. Multivariate Cox proportional hazard analysis of prognostic variables in CRC patients…
View article: Supplementary Figure 7 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Supplementary Figure 7 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
IFN-β and MET inhibitors enhance apoptosis but inhibit the proliferation of CRC cells.
View article: Supplementary Figure 3 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Supplementary Figure 3 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
siMET transfection arrests proliferation and migration of CRC cells.
View article: Supplementary Figure 6 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Supplementary Figure 6 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
IC50 determination of PF-04217903 and IFN-β in LoVo and SW620 cells.
View article: Supplementary Figure 2 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Supplementary Figure 2 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
Enhanced expression of MET correlates with progression in human CRC.
View article: Supplementary Figure 4 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Supplementary Figure 4 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
siMET transfection arrests invasion of CRC cells.
View article: Supplementary Figure 5 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Supplementary Figure 5 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
BATF2 is an IFN-stimulated gene.
View article: Supplementary Figure 4 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Supplementary Figure 4 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
siMET transfection arrests invasion of CRC cells.
View article: Supplementary Figure 1 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Supplementary Figure 1 from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
BATF2 regulates targets of c-Jun/AP-1.
View article: Data from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Data from BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
Purpose: BATF2, a novel IFN-stimulated gene, inhibits tumor cell proliferation, invasion, and migration. The objectives of this study were to determine how BATF2 expression is associated with colorectal cancer progression and…
View article: Correction to: Genome‑wide identification of NRAMP family genes in Populus trichocarpa and their roles in transport of heavy metals
Correction to: Genome‑wide identification of NRAMP family genes in Populus trichocarpa and their roles in transport of heavy metals Open
View article: PTEN deletion may be associated with CD8+ T-cell exhaustion in diffuse large B-cell lymphoma
PTEN deletion may be associated with CD8+ T-cell exhaustion in diffuse large B-cell lymphoma Open
'T-cell exhaustion' is a broad term describing the response of T cells to chronic antigen stimulation, initially in chronic viral infection and then extended to tumors. By definition, whether T-cell exhaustion occurs in diffuse large B-cel…
View article: The immune checkpoint expression in the tumor immune microenvironment of DLBCL: Clinicopathologic features and prognosis
The immune checkpoint expression in the tumor immune microenvironment of DLBCL: Clinicopathologic features and prognosis Open
Background & aims The immune checkpoint recently provides a new strategy for the immunotherapy of malignant tumors. However, the role in the immune microenvironment of DLBCL is not completely clear. Methods We detected the expression o…
View article: Correction: BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs
Correction: BATF2 Deficiency Promotes Progression in Human Colorectal Cancer via Activation of HGF/MET Signaling: A Potential Rationale for Combining MET Inhibitors with IFNs Open
In the original version of this article (1), four images were duplicated: in Fig. 2F, the Mock and NC dot plots for SW620 were the same (Mock is correct); in Fig. 3A, the Mock and NC images for 0 and 12 hours were the same (Mock is correct…
View article: Screening and identification of differentially expressed microRNAs in diffuse large B‑cell lymphoma based on microRNA microarray
Screening and identification of differentially expressed microRNAs in diffuse large B‑cell lymphoma based on microRNA microarray Open
Diffuse large B-cell lymphoma (DLBCL) is the most common type of B-cell non-Hodgkin lymphoma in adults and the pathogenesis of DLBCL is multifactorial and complex. Understanding the molecular mechanisms involved in DLBCL is important to id…
View article: Association between maternal plasma ferritin concentration, iron supplement use, and the risk of gestational diabetes: a prospective cohort study
Association between maternal plasma ferritin concentration, iron supplement use, and the risk of gestational diabetes: a prospective cohort study Open
View article: Trends in Molecular Testing of Lung Cancer in Mainland People’s Republic of China Over the Decade 2010 to 2019
Trends in Molecular Testing of Lung Cancer in Mainland People’s Republic of China Over the Decade 2010 to 2019 Open