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View article: Arbeitsbeeinträchtigung bei Patienten mit axialer Spondyloarthritis in Österreich
Arbeitsbeeinträchtigung bei Patienten mit axialer Spondyloarthritis in Österreich Open
Judith Haschka, Wolfgang Kranewitter, Miriam Hucke, Richard Finsterwalder, Dirk Meyer-Olson , et al. · 2023
Zusammenfassung Hintergrund: Axiale Spondyloarthritis (axSpA) bringt für Patienten neben körperlicher und psychischer Beeinträchtigung oft eine verminderte gesundheitsbezogene Lebensqualität mit sich. Mit dem Krankheitsbeginn im erwerbsfäh…
View article: Spontaneous reshaping of vertebral fractures in an adolescent with osteogenesis imperfecta
Spontaneous reshaping of vertebral fractures in an adolescent with osteogenesis imperfecta Open
Rodrigo Montero-Lopez, Elisabeth Laurer, Katharina Tischlinger, Dóra Nagy, Mario Scala , et al. · 2022
Vertebral reshaping is explained by the stabilization of bone mineral density (BMD) and the remaining growth capacity the children. We hypothesize that spontaneous reshaping may occur in milder forms of OI, and that puberty may be a key me…
View article: Successful kidney transplantation in a patient with pre-existing chronic myeloid leukemia treated with imatinib
Successful kidney transplantation in a patient with pre-existing chronic myeloid leukemia treated with imatinib Open
Ursula Thiem, Veronika Buxhofer‐Ausch, Wolfgang Kranewitter, Gerald Webersinke, Wolfgang Enkner , et al. · 2020
Active malignancy is an absolute contraindication to kidney transplantation. As for chronic myeloid leukemia (CML), a Philadelphia chromosome-positive myeloproliferative neoplasm, the introduction of tyrosine kinase inhibitors has transfor…
View article: Overexpression of the proneural transcription factor ASCL1 in chronic lymphocytic leukemia with a t(12;14)(q23.2;q32.3)
Overexpression of the proneural transcription factor ASCL1 in chronic lymphocytic leukemia with a t(12;14)(q23.2;q32.3) Open
Theodora Malli, Melanie Rammer, Sabrina Haslinger, Jonathan Burghofer, Sonja Burgstaller , et al. · 2018
We identified ASCL1 as a novel IGH-partner gene in CLL. The neural transcription factor was strongly overexpressed in the patient's CLL cells. Microarray gene expression analysis revealed the strong upregulation of INSM1




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