Xiantao Wang
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View article: HMGB1 regulates autophagy via TLR4/NF-κB signaling pathway in myocardial injury induced by coronary microembolization
HMGB1 regulates autophagy via TLR4/NF-κB signaling pathway in myocardial injury induced by coronary microembolization Open
View article: Three-Dimensional Reconstruction of Partially Coherent Scatterers Using Iterative Sub-Network Generation Method
Three-Dimensional Reconstruction of Partially Coherent Scatterers Using Iterative Sub-Network Generation Method Open
Synthetic aperture radar tomography (TomoSAR) has gained significant attention for three-dimensional (3D) imaging in urban environments. A notable limitation of traditional TomoSAR approaches is their primary focus on persistent scatterers…
View article: An improved bridge reference network for 3-D SAR tomography based on regional growing strategy
An improved bridge reference network for 3-D SAR tomography based on regional growing strategy Open
The utilization of synthetic aperture radar tomography (TomoSAR) technology for the three-dimensional imaging of urban areas has garnered significant attention. In the case of multi-pass spaceborne TomoSAR, eliminating the atmospheric phas…
View article: Chemoproteomic capture of RNA binding activity in living cells
Chemoproteomic capture of RNA binding activity in living cells Open
View article: Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer
Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer Open
Background Heterogeneous nuclear ribonucleoprotein K (HNRNPK) regulates pre-mRNA processing and long non-coding RNA localization in the nucleus. It was previously shown that shuttling of HNRNPK to the cytoplasm promotes cell proliferation …
View article: Supplementary Figure 1 from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism
Supplementary Figure 1 from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism Open
Supplementary Figure 1. miR-450a overexpression and effects in SKOV-3 ovarian cancer cell line.
View article: Supplementary Figure 2 from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism
Supplementary Figure 2 from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism Open
Supplementary Figure 2. miR-450b similar seed sequence and effects after its overexpression.
View article: Supplementary Table 1a from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism
Supplementary Table 1a from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism Open
Supplementary Table 1a. List of potential total miRNA and miR-450a only binding sites from A2780 control cell line
View article: Supplementary Table 1a from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism
Supplementary Table 1a from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism Open
Supplementary Table 1a. List of potential total miRNA and miR-450a only binding sites from A2780 control cell line
View article: Supplementary Table 2 from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism
Supplementary Table 2 from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism Open
Supplementary Table 2. List of targets from miR-450a derived from PAR-CLIP analysis.
View article: Supplementary Table 1b from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism
Supplementary Table 1b from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism Open
Supplementary Table 1. List of potential total miRNA and miR-450a only binding sites from A2780 cell line and expressing miR-450a
View article: Supplementary Figure 2 from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism
Supplementary Figure 2 from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism Open
Supplementary Figure 2. miR-450b similar seed sequence and effects after its overexpression.
View article: Data from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism
Data from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism Open
Dysregulation of miRNA expression is associated with multiple diseases, including cancers, in which small RNAs can have either oncogenic or tumor suppressive functions. Here we investigated the potential tumor suppressive function of miR-4…
View article: Supplementary Figure 3 from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism
Supplementary Figure 3 from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism Open
Supplementary Figure 3. Protein blot from AGO proteins from nuclear and cytoplasmic portions of A2780.
View article: Data from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism
Data from miR-450a Acts as a Tumor Suppressor in Ovarian Cancer by Regulating Energy Metabolism Open
Dysregulation of miRNA expression is associated with multiple diseases, including cancers, in which small RNAs can have either oncogenic or tumor suppressive functions. Here we investigated the potential tumor suppressive function of miR-4…
View article: Data from The Role of Protein Binding in Induction of Apoptosis by Phenethyl Isothiocyanate and Sulforaphane in Human Non–Small Lung Cancer Cells
Data from The Role of Protein Binding in Induction of Apoptosis by Phenethyl Isothiocyanate and Sulforaphane in Human Non–Small Lung Cancer Cells Open
Induction of apoptosis underlies a mechanism for inhibiting tumorigenesis by phenethyl isothiocyanate (PEITC) and sulforaphane (SFN). However, the upstream events by which isothiocyanates (ITC) induce apoptosis have not been fully investig…
View article: Supplementary Methods and Materials from The Role of Protein Binding in Induction of Apoptosis by Phenethyl Isothiocyanate and Sulforaphane in Human Non–Small Lung Cancer Cells
Supplementary Methods and Materials from The Role of Protein Binding in Induction of Apoptosis by Phenethyl Isothiocyanate and Sulforaphane in Human Non–Small Lung Cancer Cells Open
Supplementary Methods and Materials from The Role of Protein Binding in Induction of Apoptosis by Phenethyl Isothiocyanate and Sulforaphane in Human Non–Small Lung Cancer Cells
View article: Data from The Role of Protein Binding in Induction of Apoptosis by Phenethyl Isothiocyanate and Sulforaphane in Human Non–Small Lung Cancer Cells
Data from The Role of Protein Binding in Induction of Apoptosis by Phenethyl Isothiocyanate and Sulforaphane in Human Non–Small Lung Cancer Cells Open
Induction of apoptosis underlies a mechanism for inhibiting tumorigenesis by phenethyl isothiocyanate (PEITC) and sulforaphane (SFN). However, the upstream events by which isothiocyanates (ITC) induce apoptosis have not been fully investig…
View article: Supplementary Methods and Materials from The Role of Protein Binding in Induction of Apoptosis by Phenethyl Isothiocyanate and Sulforaphane in Human Non–Small Lung Cancer Cells
Supplementary Methods and Materials from The Role of Protein Binding in Induction of Apoptosis by Phenethyl Isothiocyanate and Sulforaphane in Human Non–Small Lung Cancer Cells Open
Supplementary Methods and Materials from The Role of Protein Binding in Induction of Apoptosis by Phenethyl Isothiocyanate and Sulforaphane in Human Non–Small Lung Cancer Cells
View article: Generation of a TBX20-knockout human embryonic stem line by CRISPR/Cas9 system
Generation of a TBX20-knockout human embryonic stem line by CRISPR/Cas9 system Open
The TBX20 gene plays a crucial role in embryonic development and has been involved in various diseases, such as heart defects, intellectual disability, and cancer. Herein, we have established a TBX20-knockout human embryonic stem cell line…
View article: Additional file 1 of Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer
Additional file 1 of Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer Open
Supplementary Material 1
View article: Additional file 2 of Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer
Additional file 2 of Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer Open
Supplementary Material 2
View article: Additional file 7 of Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer
Additional file 7 of Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer Open
Supplementary Material 7
View article: Additional file 3 of Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer
Additional file 3 of Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer Open
Supplementary Material 3
View article: Additional file 8 of Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer
Additional file 8 of Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer Open
Supplementary Material 8
View article: Additional file 6 of Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer
Additional file 6 of Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer Open
Supplementary Material 6
View article: Additional file 4 of Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer
Additional file 4 of Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer Open
Supplementary Material 4
View article: Additional file 5 of Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer
Additional file 5 of Keratin 19 binds and regulates cytoplasmic HNRNPK mRNA targets in triple-negative breast cancer Open
Supplementary Material 5
View article: Mitogen-activated protein kinase phosphatase-1 controls PD-L1 expression by regulating type I interferon during systemic Escherichia coli infection
Mitogen-activated protein kinase phosphatase-1 controls PD-L1 expression by regulating type I interferon during systemic Escherichia coli infection Open
Mitogen-activated protein kinase phosphatase 1 (Mkp-1) KO mice produce elevated cytokines and exhibit increased mortality and bacterial burden following systemic Escherichia coli infection. To understand how Mkp-1 affects immune defense, w…
View article: HNRNPK is retained in the cytoplasm by Keratin 19 to stabilize target mRNAs
HNRNPK is retained in the cytoplasm by Keratin 19 to stabilize target mRNAs Open
Heterogeneous nuclear ribonucleoprotein K (HNRNPK) regulates pre-mRNA processing and long non-coding RNA localization in the nucleus. It was previously shown that shuttling of HNRNPK to the cytoplasm promotes cell proliferation and cancer …