Yongzhe Che
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View article: Supplementary Figure 3 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
Supplementary Figure 3 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression Open
Supplementary Figure 3 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8+ T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
View article: Supplementary Figures 1-7 from Antigen Presentation by Dendritic Cells in Tumors Is Disrupted by Altered Metabolism that Involves Pyruvate Kinase M2 and Its Interaction with SOCS3
Supplementary Figures 1-7 from Antigen Presentation by Dendritic Cells in Tumors Is Disrupted by Altered Metabolism that Involves Pyruvate Kinase M2 and Its Interaction with SOCS3 Open
Supplementary Figures 1-7 from Antigen Presentation by Dendritic Cells in Tumors Is Disrupted by Altered Metabolism that Involves Pyruvate Kinase M2 and Its Interaction with SOCS3
View article: Data from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
Data from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression Open
Suppressor of cytokine signaling 3 (SOCS3) expression in bone marrow cells (BMC) was up-regulated upon exposure to interleukin 6, lipopolysaccharide, or tumor-associated factors. But, how the up-regulated SOCS3 affects differentiation of B…
View article: Supplementary Figure 4 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
Supplementary Figure 4 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression Open
Supplementary Figure 4 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8+ T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
View article: Supplementary Figure 1 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
Supplementary Figure 1 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression Open
Supplementary Figure 1 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8+ T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
View article: Supplementary Table 1 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
Supplementary Table 1 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression Open
Supplementary Table 1 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8+ T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
View article: Data from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
Data from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression Open
Suppressor of cytokine signaling 3 (SOCS3) expression in bone marrow cells (BMC) was up-regulated upon exposure to interleukin 6, lipopolysaccharide, or tumor-associated factors. But, how the up-regulated SOCS3 affects differentiation of B…
View article: Supplementary Table 1 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
Supplementary Table 1 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression Open
Supplementary Table 1 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8+ T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
View article: Supplementary Figure 6 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
Supplementary Figure 6 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression Open
Supplementary Figure 6 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8+ T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
View article: Supplementary Figure 1 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
Supplementary Figure 1 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression Open
Supplementary Figure 1 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8+ T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
View article: Supplementary Figure 2 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
Supplementary Figure 2 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression Open
Supplementary Figure 2 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8+ T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
View article: Data from Antigen Presentation by Dendritic Cells in Tumors Is Disrupted by Altered Metabolism that Involves Pyruvate Kinase M2 and Its Interaction with SOCS3
Data from Antigen Presentation by Dendritic Cells in Tumors Is Disrupted by Altered Metabolism that Involves Pyruvate Kinase M2 and Its Interaction with SOCS3 Open
Dendritic cell (DC) function is negatively affected by tumors and tumor-derived factors, but little is known about the underlying mechanisms. Here, we show that intracellular SOCS3 in DCs binds to pyruvate kinase type M2 (M2-PK), which pla…
View article: Supplementary Figures 1-7 from Antigen Presentation by Dendritic Cells in Tumors Is Disrupted by Altered Metabolism that Involves Pyruvate Kinase M2 and Its Interaction with SOCS3
Supplementary Figures 1-7 from Antigen Presentation by Dendritic Cells in Tumors Is Disrupted by Altered Metabolism that Involves Pyruvate Kinase M2 and Its Interaction with SOCS3 Open
Supplementary Figures 1-7 from Antigen Presentation by Dendritic Cells in Tumors Is Disrupted by Altered Metabolism that Involves Pyruvate Kinase M2 and Its Interaction with SOCS3
View article: Supplementary Figure 2 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
Supplementary Figure 2 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression Open
Supplementary Figure 2 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8+ T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
View article: Supplementary Figure 6 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
Supplementary Figure 6 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression Open
Supplementary Figure 6 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8+ T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
View article: Supplementary Figure 5 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
Supplementary Figure 5 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression Open
Supplementary Figure 5 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8+ T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
View article: Supplementary Figure 3 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
Supplementary Figure 3 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression Open
Supplementary Figure 3 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8+ T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
View article: Data from Antigen Presentation by Dendritic Cells in Tumors Is Disrupted by Altered Metabolism that Involves Pyruvate Kinase M2 and Its Interaction with SOCS3
Data from Antigen Presentation by Dendritic Cells in Tumors Is Disrupted by Altered Metabolism that Involves Pyruvate Kinase M2 and Its Interaction with SOCS3 Open
Dendritic cell (DC) function is negatively affected by tumors and tumor-derived factors, but little is known about the underlying mechanisms. Here, we show that intracellular SOCS3 in DCs binds to pyruvate kinase type M2 (M2-PK), which pla…
View article: Supplementary Figure 5 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
Supplementary Figure 5 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression Open
Supplementary Figure 5 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8+ T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
View article: Supplementary Figure 4 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
Supplementary Figure 4 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8<sup>+</sup> T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression Open
Supplementary Figure 4 from Suppressor of Cytokine Signaling 3 Promotes Bone Marrow Cells to Differentiate into CD8+ T Lymphocytes in Lung Tissue via Up-Regulating Notch1 Expression
View article: An Injectable Dual‐Function Hydrogel Protects Against Myocardial Ischemia/Reperfusion Injury by Modulating ROS/NO Disequilibrium
An Injectable Dual‐Function Hydrogel Protects Against Myocardial Ischemia/Reperfusion Injury by Modulating ROS/NO Disequilibrium Open
Acute myocardial infarction (MI) is the leading cause of death worldwide. Exogenous delivery of nitric oxide (NO) to the infarcted myocardium has proven to be an effective strategy for treating MI due to the multiple physiological function…
View article: Mechanically reinforced biotubes for arterial replacement and arteriovenous grafting inspired by architectural engineering
Mechanically reinforced biotubes for arterial replacement and arteriovenous grafting inspired by architectural engineering Open
There is a lack in clinically-suitable vascular grafts. Biotubes, prepared using in vivo tissue engineering, show potential for vascular regeneration. However, their mechanical strength is typically poor. Inspired by architectural design o…
View article: Loss of the voltage-gated proton channel Hv1 decreases insulin secretion and leads to hyperglycemia and glucose intolerance in mice
Loss of the voltage-gated proton channel Hv1 decreases insulin secretion and leads to hyperglycemia and glucose intolerance in mice Open
Insulin secretion by pancreatic islet β-cells is regulated by glucose levels and is accompanied by proton generation. The voltage-gated proton channel Hv1 is present in pancreatic β-cells and extremely selective for protons. However, wheth…
View article: Lactobacillus maintains healthy gut mucosa by producing L-Ornithine
Lactobacillus maintains healthy gut mucosa by producing L-Ornithine Open
Gut mucosal layers are crucial in maintaining the gut barrier function. Gut microbiota regulate homeostasis of gut mucosal layer via gut immune cells such as RORγt (+) IL-22(+) ILC3 cells, which can influence the proliferation of mucosal c…
View article: Embryonic lethality in mice lacking Trim59 due to impaired gastrulation development
Embryonic lethality in mice lacking Trim59 due to impaired gastrulation development Open
TRIM family members have been implicated in a variety of biological processes such as differentiation and development. We here found that Trim59 plays a critical role in early embryo development from blastocyst stage to gastrula. There exi…
View article: Prostaglandin E<sub>2</sub> hydrogel improves cutaneous wound healing via M2 macrophages polarization
Prostaglandin E<sub>2</sub> hydrogel improves cutaneous wound healing via M2 macrophages polarization Open
Wound healing is regulated by a complex series of events and overlapping phases. A delicate balance of cytokines and mediators in tissue repair is required for optimal therapy in clinical applications. Molecular imaging technologies, with …
View article: Gut REG3γ-Associated Lactobacillus Induces Anti-inflammatory Macrophages to Maintain Adipose Tissue Homeostasis
Gut REG3γ-Associated Lactobacillus Induces Anti-inflammatory Macrophages to Maintain Adipose Tissue Homeostasis Open
Gut microbiota may not only affect composition of local immune cells but also affect systemic immune cells. However, it is not completely clear how gut microbiota modulate these immune systems. Here, we found that there exist expanded macr…
View article: Embryonic lethality in mice lacking Trim59 due to impaired gastrulation development
Embryonic lethality in mice lacking Trim59 due to impaired gastrulation development Open
TRIM family members have been implicated in a variety of biological processes such as differentiation and development. We here found that Trim59 plays a critical role in early embryo development from blastocyst stage to gastrula. There exi…
View article: Herbal formula Xian-Fang-Huo-Ming-Yin regulates differentiation of lymphocytes and production of pro-inflammatory cytokines in collagen-induced arthritis mice
Herbal formula Xian-Fang-Huo-Ming-Yin regulates differentiation of lymphocytes and production of pro-inflammatory cytokines in collagen-induced arthritis mice Open
XFHM can regulate and maintain the immunologic balance of lymphocytic immunity and inhibit the production of pro-inflammatory cytokines, thus suppressing the pathological changes of RA. Therefore, XFHM may be used as an application of trad…