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View article: Seasonally Intensified Mud Shrimp Bioturbation Hinders Seagrass Restoration
Seasonally Intensified Mud Shrimp Bioturbation Hinders Seagrass Restoration Open
Understanding how disturbances affect marine foundation species is critical for enhancing the success of coastal ecosystem restoration. Extreme bioturbation by burrowing animals is increasingly impacting coastal vegetated habitats worldwid…
View article: Uncovering microbial interactions in a persistent Planktothrix bloom: Towards early biomarker identification in hypereutrophic lakes
Uncovering microbial interactions in a persistent Planktothrix bloom: Towards early biomarker identification in hypereutrophic lakes Open
Cyanobacterial harmful algal blooms pose significant threats to global water supplies, ecosystems, and economies. Among the harmful cyanobacteria, Planktothrix, a resilient and toxin-producing filamentous cyanobacterium, has garnered incre…
View article: Release of algal organic matter from cyanobacteria following application of USEPA-registered chemical algaecides
Release of algal organic matter from cyanobacteria following application of USEPA-registered chemical algaecides Open
Increased occurrence of harmful algal blooms significantly impedes uses of freshwater resources, especially as potable water supply. Rapid mitigation using algaecides is common; however, the potential release of algal organic matter (AOM) …
View article: Impact of harmful algal bloom severity on bacterial communities in a full-scale biological filtration system for drinking water treatment
Impact of harmful algal bloom severity on bacterial communities in a full-scale biological filtration system for drinking water treatment Open
The occurrence of harmful algal blooms (HABs) in freshwater environments has been expanded worldwide with growing frequency and severity. HABs can pose a threat to public water supplies, raising concerns about safety of treated water. Many…
View article: Field and laboratory studies of fluorescence-based technologies for real-time tracking of cyanobacterial cell lysis and potential microcystins release
Field and laboratory studies of fluorescence-based technologies for real-time tracking of cyanobacterial cell lysis and potential microcystins release Open
Elevated levels of dissolved microcystins (MCs) in source water due to rapid cell lysis of harmful cyanobacterial blooms may pose serious challenges for drinking water treatment. Catastrophic cell lysis can result from outbreaks of natural…
View article: Supplementary Figure 8 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Figure 8 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 662K, Figure 8. Higher expression levels of KITENIN affect the survival of CRC cells to the anti-proliferative effects of cetuximab.
View article: Supplementary Figure 7 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Figure 7 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 138K, Figure 7. Co-transfection of both KITENIN and ErbB4 induced greater elevation in AP-1 activity after EGF treatment.
View article: Supplementary Figure 6 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Figure 6 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 206K, Figure 6. Modulation of multiple RTKs in KITENIN-overexpressed Caco2 or KITENIN-knockdown HCT116 CRC cells.
View article: Data from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Data from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
Purpose: EGF-stimulated signaling via EGF receptor (EGFR) is important in colorectal tumorigenesis and drug targeting. However, anti-EGFR therapy is not effective in a subset of patients with colorectal cancer, suggesting that unidentified…
View article: Data from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Data from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
Purpose: EGF-stimulated signaling via EGF receptor (EGFR) is important in colorectal tumorigenesis and drug targeting. However, anti-EGFR therapy is not effective in a subset of patients with colorectal cancer, suggesting that unidentified…
View article: Supplementary Methods and Figure Legends from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Methods and Figure Legends from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 456K
View article: Supplementary Figure 4 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Figure 4 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 158K, Figure 4. Effects of Dvl2-knockdown on c-Jun and of EGF on c-Jun/Dvl2 level in KITENIN-transfected cells.
View article: Supplementary Figure 7 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Figure 7 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 138K, Figure 7. Co-transfection of both KITENIN and ErbB4 induced greater elevation in AP-1 activity after EGF treatment.
View article: Supplementary Figure 4 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Figure 4 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 158K, Figure 4. Effects of Dvl2-knockdown on c-Jun and of EGF on c-Jun/Dvl2 level in KITENIN-transfected cells.
View article: Supplementary Figure 6 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Figure 6 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 206K, Figure 6. Modulation of multiple RTKs in KITENIN-overexpressed Caco2 or KITENIN-knockdown HCT116 CRC cells.
View article: Supplementary Figure 3 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Figure 3 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 135K, Figure 3. EGF-induced AP-1 synergy under elevated KITENIN conditions does not require activated JNK.
View article: Supplementary Figure 2 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Figure 2 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 177K, Figure 2. Effects of functional blockade of EGFR via treatment of an anti-EGFR monoclonal antibody on enhanced cell invasion by EGF.
View article: Supplementary Figure 3 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Figure 3 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 135K, Figure 3. EGF-induced AP-1 synergy under elevated KITENIN conditions does not require activated JNK.
View article: Supplementary Figure 2 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Figure 2 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 177K, Figure 2. Effects of functional blockade of EGFR via treatment of an anti-EGFR monoclonal antibody on enhanced cell invasion by EGF.
View article: Supplementary Figure 5 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Figure 5 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 143K, Figure 5. Dvl2-knockdown does not increase transcription of c-Jun and Dvl2 does not interact with c-Jun.
View article: Supplementary Figure 5 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Figure 5 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 143K, Figure 5. Dvl2-knockdown does not increase transcription of c-Jun and Dvl2 does not interact with c-Jun.
View article: Supplementary Figure 8 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Figure 8 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 662K, Figure 8. Higher expression levels of KITENIN affect the survival of CRC cells to the anti-proliferative effects of cetuximab.
View article: Supplementary Figure 1 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Figure 1 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 126K, Figure 1. EGF induces synergic AP-1 activation under elevated KITENIN in an EGFR- and EGFR-kinase-independent manner.
View article: Supplementary Methods and Figure Legends from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Methods and Figure Legends from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 456K
View article: Supplementary Figure 1 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells
Supplementary Figure 1 from An Unconventional KITENIN/ErbB4-Mediated Downstream Signal of EGF Upregulates c-Jun and the Invasiveness of Colorectal Cancer Cells Open
PDF file - 126K, Figure 1. EGF induces synergic AP-1 activation under elevated KITENIN in an EGFR- and EGFR-kinase-independent manner.
View article: Supplementary Figure 4 from The Cell Death–Inducing Activity of the Peptide Containing Noxa Mitochondrial-Targeting Domain Is Associated with Calcium Release
Supplementary Figure 4 from The Cell Death–Inducing Activity of the Peptide Containing Noxa Mitochondrial-Targeting Domain Is Associated with Calcium Release Open
Supplementary Figure 4 from The Cell Death–Inducing Activity of the Peptide Containing Noxa Mitochondrial-Targeting Domain Is Associated with Calcium Release
View article: Supplementary Methods, Figure Legends 1-4 from The Cell Death–Inducing Activity of the Peptide Containing Noxa Mitochondrial-Targeting Domain Is Associated with Calcium Release
Supplementary Methods, Figure Legends 1-4 from The Cell Death–Inducing Activity of the Peptide Containing Noxa Mitochondrial-Targeting Domain Is Associated with Calcium Release Open
Supplementary Methods, Figure Legends 1-4 from The Cell Death–Inducing Activity of the Peptide Containing Noxa Mitochondrial-Targeting Domain Is Associated with Calcium Release
View article: Supplementary Methods, Figure Legends 1-4 from The Cell Death–Inducing Activity of the Peptide Containing Noxa Mitochondrial-Targeting Domain Is Associated with Calcium Release
Supplementary Methods, Figure Legends 1-4 from The Cell Death–Inducing Activity of the Peptide Containing Noxa Mitochondrial-Targeting Domain Is Associated with Calcium Release Open
Supplementary Methods, Figure Legends 1-4 from The Cell Death–Inducing Activity of the Peptide Containing Noxa Mitochondrial-Targeting Domain Is Associated with Calcium Release
View article: Supplementary Figure 3 from The Cell Death–Inducing Activity of the Peptide Containing Noxa Mitochondrial-Targeting Domain Is Associated with Calcium Release
Supplementary Figure 3 from The Cell Death–Inducing Activity of the Peptide Containing Noxa Mitochondrial-Targeting Domain Is Associated with Calcium Release Open
Supplementary Figure 3 from The Cell Death–Inducing Activity of the Peptide Containing Noxa Mitochondrial-Targeting Domain Is Associated with Calcium Release