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Supplementary Table 2 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines Open

Helena Shaked, Idit Shiff, Miriam Kott-Gutkowski, Zahava Siegfried, Ygal Haupt , et al. · 2023

Supplementary Table 2 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines

Data from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines Open

Helena Shaked, Idit Shiff, Miriam Kott-Gutkowski, Zahava Siegfried, Ygal Haupt , et al. · 2023

The p53 tumor suppressor protein is a transcription factor that plays a key role in the cellular response to stress and cancer prevention. Upon activation, p53 regulates a large variety of genes causing cell cycle arrest, apoptosis, or sen…

Supplementary Table 1 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines Open

Helena Shaked, Idit Shiff, Miriam Kott-Gutkowski, Zahava Siegfried, Ygal Haupt , et al. · 2023

Supplementary Table 1 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines

Supplementary Figure 2 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines Open

Helena Shaked, Idit Shiff, Miriam Kott-Gutkowski, Zahava Siegfried, Ygal Haupt , et al. · 2023

Supplementary Figure 2 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines

Data from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines Open

Helena Shaked, Idit Shiff, Miriam Kott-Gutkowski, Zahava Siegfried, Ygal Haupt , et al. · 2023

The p53 tumor suppressor protein is a transcription factor that plays a key role in the cellular response to stress and cancer prevention. Upon activation, p53 regulates a large variety of genes causing cell cycle arrest, apoptosis, or sen…

Supplementary Table 2 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines Open

Helena Shaked, Idit Shiff, Miriam Kott-Gutkowski, Zahava Siegfried, Ygal Haupt , et al. · 2023

Supplementary Table 2 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines

Supplementary Figure 2 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines Open

Helena Shaked, Idit Shiff, Miriam Kott-Gutkowski, Zahava Siegfried, Ygal Haupt , et al. · 2023

Supplementary Figure 2 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines

Supplementary Figure 3 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines Open

Helena Shaked, Idit Shiff, Miriam Kott-Gutkowski, Zahava Siegfried, Ygal Haupt , et al. · 2023

Supplementary Figure 3 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines

Supplementary Figure 3 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines Open

Helena Shaked, Idit Shiff, Miriam Kott-Gutkowski, Zahava Siegfried, Ygal Haupt , et al. · 2023

Supplementary Figure 3 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines

Supplementary Figure 4 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines Open

Helena Shaked, Idit Shiff, Miriam Kott-Gutkowski, Zahava Siegfried, Ygal Haupt , et al. · 2023

Supplementary Figure 4 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines

Supplementary Figure 4 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines Open

Helena Shaked, Idit Shiff, Miriam Kott-Gutkowski, Zahava Siegfried, Ygal Haupt , et al. · 2023

Supplementary Figure 4 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines

Supplementary Table 3 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines Open

Helena Shaked, Idit Shiff, Miriam Kott-Gutkowski, Zahava Siegfried, Ygal Haupt , et al. · 2023

Supplementary Table 3 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines

Supplementary Figure 1 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines Open

Helena Shaked, Idit Shiff, Miriam Kott-Gutkowski, Zahava Siegfried, Ygal Haupt , et al. · 2023

Supplementary Figure 1 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines

Supplementary Figure 1 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines Open

Helena Shaked, Idit Shiff, Miriam Kott-Gutkowski, Zahava Siegfried, Ygal Haupt , et al. · 2023

Supplementary Figure 1 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines

Supplementary Table 1 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines Open

Helena Shaked, Idit Shiff, Miriam Kott-Gutkowski, Zahava Siegfried, Ygal Haupt , et al. · 2023

Supplementary Table 1 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines

Supplementary Table 3 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines Open

Helena Shaked, Idit Shiff, Miriam Kott-Gutkowski, Zahava Siegfried, Ygal Haupt , et al. · 2023

Supplementary Table 3 from Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines

RBFOX2 modulates a metastatic signature of alternative splicing in pancreatic cancer Open

Amina Jbara, Kuan‐Ting Lin, Chani Stossel, Zahava Siegfried, Haya Shqerat , et al. · 2023

Pancreatic ductal adenocarcinoma (PDA) is characterized by aggressive local invasion and metastatic spread, leading to high lethality. Although driver gene mutations during PDA progression are conserved, no specific mutation is correlated …

Targeting splicing factors for cancer therapy Open

Ariel Bashari, Zahava Siegfried, Rotem Karni · 2023

Alternative splicing (AS) of mRNAs is an essential regulatory mechanism in eukaryotic gene expression. AS misregulation, caused by either dysregulation or mutation of splicing factors, has been shown to be involved in cancer development an…

S6K1 phosphorylates Cdk1 and MSH6 to regulate DNA repair Open

Adi Amar-Schwartz, Vered Ben Hur, Amina Jbara, Yuval Cohen, Georgina D. Barnabas , et al. · 2022

The mTORC1 substrate, S6 Kinase 1 (S6K1), is involved in the regulation of cell growth, ribosome biogenesis, glucose homeostasis, and adipogenesis. Accumulating evidence has suggested a role for mTORC1 signaling in the DNA damage response.…

Author response: S6K1 phosphorylates Cdk1 and MSH6 to regulate DNA repair Open

Adi Amar-Schwartz, Vered Ben Hur, Amina Jbara, Yuval Cohen, Georgina D. Barnabas , et al. · 2022

Article Figures and data Abstract Editor's evaluation eLife digest Introduction Results Discussion Materials and methods Data availability References Decision letter Author response Article and author information Metrics Abstract The mTORC…

S6K1 phosphorylates Cdk1 and MSH6 to regulate DNA repair Open

Adi Amar-Schwartz, Vered Ben-Hur, Amina Jbara, Yuval Cohen, Georgina D. Barnabas , et al. · 2022

The mTORC1 substrate, S6 Kinase 1 (S6K1), is involved in the regulation of cell growth, ribosome biogenesis, glucose homeostasis and adipogenesis. Accumulating evidence has suggested a role for mTORC1 signaling in the DNA damage response. …

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